I wanna revamp this.
If it takes awhile for hyperplasia to happen,
what are these "quicker results" people talking about.
Fat loss? Fuller looking muscle ( insulin nutrient shuttling)
Or actual growth?
quote from a past post:
"With igf-1lr3 I think the problem is that it is very similar to a short acting insulin when it comes to its short-term effects. When a person is talking about how they didn't see much from it they are often referring to 1 or 2 short runs with it- i.e. not talking about hyperplasia results which are said to take a long time to manifest. So, what's being discussed are more along the lines of the mass gains one sees from humalog or humalin-r. I don't even want to get into the debate about long-term use and hyperplasia with igf-1lr3 because I don't have a real answer and don't know how anyone does (I mean, how would a person really measure that vs. hypertrophy? Are people doing biopsies on themselves and counting the "new" muscle cells vs hypertrophied cells...?).
So, if people are going by the actual "insulin-like" properties of the drug then, yes, it is very much about timing, carb intake etc. For me, I don't see much from even humalog if I'm not on aas. This is a very important factor. For whatever reason I just cannot seem to get the same effects from insulin when I'm not "on." I really do believe there is some benefit to igf-1 lr3 and peg-mgf. But, I can't prove it. A lot of people are going to perceive that they are getting more bang for their buck from test and insulin as it's hard to really verify the hyperplasia results and the "insulin-like" results are appear to work very similarly to actual insulin."
If it takes awhile for hyperplasia to happen,
what are these "quicker results" people talking about.
Fat loss? Fuller looking muscle ( insulin nutrient shuttling)
Or actual growth?
quote from a past post:
"With igf-1lr3 I think the problem is that it is very similar to a short acting insulin when it comes to its short-term effects. When a person is talking about how they didn't see much from it they are often referring to 1 or 2 short runs with it- i.e. not talking about hyperplasia results which are said to take a long time to manifest. So, what's being discussed are more along the lines of the mass gains one sees from humalog or humalin-r. I don't even want to get into the debate about long-term use and hyperplasia with igf-1lr3 because I don't have a real answer and don't know how anyone does (I mean, how would a person really measure that vs. hypertrophy? Are people doing biopsies on themselves and counting the "new" muscle cells vs hypertrophied cells...?).
So, if people are going by the actual "insulin-like" properties of the drug then, yes, it is very much about timing, carb intake etc. For me, I don't see much from even humalog if I'm not on aas. This is a very important factor. For whatever reason I just cannot seem to get the same effects from insulin when I'm not "on." I really do believe there is some benefit to igf-1 lr3 and peg-mgf. But, I can't prove it. A lot of people are going to perceive that they are getting more bang for their buck from test and insulin as it's hard to really verify the hyperplasia results and the "insulin-like" results are appear to work very similarly to actual insulin."