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Can you explain this please.
It just what I've read, Stewie. Perhaps you can be of help with his issue.
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Last edited:
Can you explain this please.
It just what I've read, Stewie. Perhaps you can be of help with his issue.
Sent from my iPhone using Tapatalk
To add to my last statement. After the binding occurs with two different AAS in the androgen receptors, the receptors then form dimers, or conjunctions, then it's these dimers that work to increase transcription of DNA. This results in a synergistic effect.
Hopefully this makes sense?
100-150 a week?after 30 even small amount of tren gave me wicked gyno. tried it maybe 4 times since then and all the same. lol
prami is a funny drug to use and can have many side effects, in all probability just from a quick glance you may need more help against the tren.
try switching to caber or lowering or dropping the tren.
I used to run 700mgs form kits no problem, last year I tested some tren base and had to cut it back from 150ish mgs to 100ish mgs and that was just barley tolerable...
I absolutely could agree with you on this. Ive always responded real quick to aas. And seen better results than friends on the same amount. But my body seems to adjust really fast too. Shbg, aromatizing, etc. Question for me now is figuring out how its trying to compensate so i can fight that.... Yes. And is more helpful information in short than I've read at length. Which brings more questions.
Is there a point to which saturation occurs? If so, is it defendant upon the how many receptors the body has... A while back I read an article describing a test that can be performed to determine the amount of receptors in the body, describing a ratio from 6-36 as it's scale. The writer suggests the lower the number, the greater a probability of being a "high responder" to AAS and could account for greater "sides" as opposed to someone with with more receptors whom could absorb more (thus the need for a higher dose to effect the same as the next who does less with equal response). I think it was called a GAC profile I've looked and googled for this. Can't find it, though I remember it was posted on Meso-RX (thinksteroids.com if that helps).
Or do dimers continue to be created as needed when more anabolic is introduced? And could this effect be responsible for the OP's gyno...
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... Yes. And is more helpful information in short than I've read at length. Which brings more questions.
Is there a point to which saturation occurs? If so, is it defendant upon the how many receptors the body has... A while back I read an article describing a test that can be performed to determine the amount of receptors in the body, describing a ratio from 6-36 as it's scale. The writer suggests the lower the number, the greater a probability of being a "high responder" to AAS and could account for greater "sides" as opposed to someone with with more receptors whom could absorb more (thus the need for a higher dose to effect the same as the next who does less with equal response). I think it was called a GAC profile I've looked and googled for this. Can't find it, though I remember it was posted on Meso-RX (thinksteroids.com if that helps).
Or do dimers continue to be created as needed when more anabolic is introduced? And could this effect be responsible for the OP's gyno...
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Well i have a slightly elevated psa, hence the dutasteride, so i really limit my dht derivatives..
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