Armidex causing nipple pain??

[thewarrior23]

Hey guys, just need a little help to stop my worrying I hope. I recently got some BW done to check my estrodial levels on cycle, and they were at 30pg/ml. According to bloodwork normal is 0-50pg. So I did some more research and it turns out "for men" normal estradio levels are 20-75pg/ml. It seems like I got some underdosed Aromasin and that why my estro levels were not as low as I liked. I quickly switched to adex at 1mg ED, as im a gyno parnoid guy. Got BW done a week later, and my estradiol came back at 22pg/ml, so almost a 1/3 lower.

My question is, at this level gyno should be of no concern right??

Also, this is the first time I have ran adex before. Its strrange im on almost nothing right now, no progestin related stuff. BUt Like i said my estro levels are at 22pg/ml which according to some reseach is almost borderline to low.

Well my concern is My right nipple seems to be getting tender or has been since I started the adex, but pretty much nothing on my left nipple. I read about adex and it says in some it can cause chest or breast pain, as a side, so this is what im hoping this is.

Has anyone experienced this w/ adex?? As i said im gyno paranoid so any help would be great.

 

[thewarrior23]

bump

 

[VitoBrata]

I don't think you have a whole lot to worry about. You said you on "almost nothing" what are you on?

 

[Researchstop]

I don't know where you got your Aromasin from but I don't think I would conclude that it was underdosed. I would also not use your E2 levels within normal range to determine whether or not you're going to get gyno. There are other causes of gyno. Arimidex is better at sulphatase/estrone reduction in men than E2. The small difference in your E2 levels while on Test can easily be attributed to other factors. Even what you ate before testing. If you saw a significant change between the two it would be 20+pg/ml not 8.
I guess I am saying, you're right, you're paranoid about gyno, And you're overthinking this.

 

[VitoBrata]

I don't know where you got your Aromasin from but I don't think I would conclude that it was underdosed. I would also not use your E2 levels within normal range to determine whether or not you're going to get gyno. There are other causes of gyno. Arimidex is better at sulphatase/estrone reduction in men than E2. The small difference in your E2 levels while on Test can easily be attributed to other factors. Even what you ate before testing. If you saw a significant change between the two it would be 20+pg/ml not 8.
I guess I am saying, you're right, you're paranoid about gyno, And you're overthinking this.

There you go right from an authority on the subject, you are paranoid.

 

[thewarrior23]

I just thought or read E2 levels are the strongest kind of estro in the body. Im not running anything progestin related so, i thought if i kept the big e2 levels low i should be good to go. I mean I was on aromasin and was at 30pg switched to adex and went to 22 in a week.

So at 22pg estro realted gyno is virutally impossible right??

 

[Macro]

I just thought or read E2 levels are the strongest kind of estro in the body. Im not running anything progestin related so, i thought if i kept the big e2 levels low i should be good to go. I mean I was on aromasin and was at 30pg switched to adex and went to 22 in a week.

So at 22pg estro realted gyno is virutally impossible right??

no. under the right conditions very low estrogen levels can still induce proliferation.

what exactly are you taking cycle/ancillary wise?
meds?
supplements?

 

[Macro]

as a note- how long you have been on cycle will affect results as well, suicidal inhibitors like aromasin may not produce as great a suppression of E2 in the beginning, because of the estrone sulfate(E1S) pool (which varies in size). Also in high glandular estrone producers, unless suppressed, suicidal inhibitors may still yield lower suppression (not particularly common to fall into such a category, but these people will tend to prefer dex or letrozole more often than the general populace)

 

[thewarrior23]

What proliferation???

On only like 200mg test wkly. And 1mg Adex ED. Basically HRT for myself.

So like I said shouldnt gyno be almost impossible being that my E2 levels are pretty low and Im not running and GH or progestin stuff?? Just trying to understand why even with low estro levels my right nipple is sensitive/painful. I have read that adex can cause breast pain, but will wait for more responces. Like i said I know im gyno paranoid, but would rather be safe then sorry.

Any help much appreciated fellas. Talked to some ppl and they said that just flucuationg hormones levels from an AI can cause some gyno symptons, but like i said at this point is only my right nipple thank goodness.

Any more help would be appreciated.

 

[Macro]

you are not taking any other medications, health or sports supplements, fat burners, etc?

 

[617ricky]

as a note- how long you have been on cycle will affect results as well, suicidal inhibitors like aromasin may not produce as great a suppression of E2 in the beginning, because of the estrone sulfate(E1S) pool (which varies in size). Also in high glandular estrone producers, unless suppressed, suicidal inhibitors may still yield lower suppression (not particularly common to fall into such a category, but these people will tend to prefer dex or letrozole more often than the general populace)

What does this mean? I always hear on here that Aromasin is totally superior to Arimidex - so in what cases is this not true?

 

[thewarrior23]

no other meds. If u want beter gyno protection is aromasin better than adex?

 

[Macro]

no other meds. If u want beter gyno protection is aromasin better than adex?

depends on user, but generally yes.

just as letrozole is generally the strongest of the three.

 

[Macro]

What does this mean? I always hear on here that Aromasin is totally superior to Arimidex - so in what cases is this not true?

in high glandular aromatase (testicular and adrenal, perhaps other) producers, competitive inhibitors may be more effective. this does not appear to be common, at least from general users response rates to these compounds. the majority of E is peripherally produced, and thus suicidal suppression is more effective.