U said that that protocol would not be effective at all... I dispute that claim...that's all...as for studies n what not...what is your source?
You dispute it yet make no comments as to why, you just dispute it..
It is
good you are disputing my comments, but you should dispute and question ANY other comments that are not backed up from science. I am here to just help, not targeting anyone.
When i say not effective at all this is what i mean.
Not effective is not the same as not providing results, it is not effective when it comes to cost, it is not effective when it comes to synergistic effect, it is not effective when it comes to dosing timing, it is not effective when it comes to natural MGF production. I can explain all with science. I have done so in other posts.
My source are studies, which studies? either ones i look up on pubmed, science direct, or the ones Dat provides, i do not have time to go looking up many, Dat has a amazing understanding and is able to pick the important parts out. So thats where i learn from. You ?
Example why MGF is better than any other IGF for
repair / proliferation and why if your not careful and just following others who have no idea,
how things can be dangerous.
"The fact that IGF-1 can act either as a hormone or as a local factor has complicated the analysis and interpretation of IGF-1 function, as well as its therapeutic application in different disease models. At the structural level, IGF-1 shares a 50% amino acid similarity with insulin (reviewed in Ref. [1]) but unlike the insulin gene, the IGF-1 gene encodes at least four different IGF-1 isoforms, originating from alternative splicing, alternative promoter usage and post-translational modification [4] and [5]. The different isoforms vary in structure and function and are referred to as circulating (class 2) and local (class 1) IGF-1 [5] and [6]. The variation in IGF-1 isoform expression probably reflects an underlying complexity of IGF-1 actions.
Circulating IGF-1 has consistently been associated with cancer [7], and many of the clinical trials using IGF-1 have been interrupted as a result of the potential tumorigenic effects. An autocrine form of IGF-1, called mechano growth factor (MGF), is rapidly activated and subsequently repressed in damaged and in exercised muscles, suggesting that MGF acts as a separate growth factor, having an important role in the regenerative capacity of senescent skeletal muscle [8]." -IGF-1, inflammation and stem cells: interactions during muscle regeneration, Foteini Mourkioti, Trends in Immunology Volume 26, Issue 10, October 2005, Pages 535–542