Again, half life of PEG vs. reg. MGF are not important. However the environment is crucial and it's what needs to be focused on. I'll do my best to explain.
1. Yes, Dat said merely injecting MGF does not cause it to act as MGF = proliferation. Agreed. However as
Seabiscuit Hogg refferenced a post (of which I am unaware of as well), exactly what is the environment MGF is introduced into? It's clear MGF only exists when damaged and torn muscle occurs. That's Post WO. Of course, injecting any other time (depending on the environment) anythings possible - no guarantee of proliferation. In fact, I'll go one better, do not expect MGF to ever act like it supposed to unless you admin it Post WO when natural/internal MGF exists!
2. The next thing is, once prolferation commences, it can continue, not contingent upon any particular peptide assistance. Meaning, once proliferation begins, the only thing I'm aware of capable of altering its expression/synthesis is the environment itself, rather than the elimination/clearance of the peptide. Thus, whether peglated or not, once its cleared our system it'll be the environment itself dictating proliferation or differentiation. We can however stop proliferation if we so choose to.
A quotation from Dat, "
exogenously administered IGF1 will interfere with MGF expression".
In a nut shell, IF proliferation (MGF expression) exists, IGF will alter snythesis into differentiation. But again, it's kicking off and then maintaining MGF expression that seems to be the issue.
Exactly! Very nice. That brings me to:
3. If MGF only occurs after a WO and we desire to promote its expression, if we are to be assured it will act like MGF, then it makes perfect sense to admin it at the only time we can expect proliferation to exist - Post WO.
Hmmm, you're link doesn't work. Though I am aware of the post you're referring to. So here it is again for anyone else wanting to see it.
http://www.professionalmuscle.com/f...hrp-6-basic-guides-post487161.html#post487161