Is aborption time always dependent on ester type? For example would Tren E and Test E become active in the bloodstream in the same amount of time? Or does compound type play a role?
Is aborption time always dependent on ester type? For example would Tren E and Test E become active in the bloodstream in the same amount of time? Or does compound type play a role?
I'd just point out that longer esters are not enzymatically harder to "break off". That's not what causes the longer half-life. Rather, longer esters are more lipophillic, so they remain in the injection depot longer. They're less likely to partition into the aqueous interstitual fluid around the injection site. Once they leave the depot though, the ester is quickly removed by esterases. The rate of that enzymatic breakdown is rapid regardless of the ester length.
Oil will slow the absorption, anything in water will enter the bloodstream quicker. Usually hurts more too, as the water is the first to leave and sometimes the hormone stays behind in the muscle causing discomfort