Is aborption time always dependent on ester type? For example would Tren E and Test E become active in the bloodstream in the same amount of time? Or does compound type play a role?
Thanks,
Showy
I'd just point out that longer esters are not enzymatically harder to "break off". That's not what causes the longer half-life. Rather, longer esters are more lipophillic, so they remain in the injection depot longer. They're less likely to partition into the aqueous interstitual fluid around the injection site. Once they leave the depot though, the ester is quickly removed by esterases. The rate of that enzymatic breakdown is rapid regardless of the ester length.the ester is what the body must 'break off' the parent molecule to use it. since the ester is the same, the rates of enzymatic breakdown are the same.