I call this the 'I can't find my own evidence, so I'll pull apart the study stated' strategy.
As you know 95% of the studies and clinical data we look at when we formulate an opinion isn't 100% relevant to the individual. Otherwise you would go claiming Tamoxifen wont work on my gynecomastia because I'm not a female with breast cancer.
How about you show me a study that confirms Mesterolone does NOT negatively impact lipids?
Answer the questions in my above post without getting sassy with an unnecessary undertone.. I'll call your response the "I'll dodge the questions with a question by making more comparisons that are irreverent to the substance just to support the I'm Swifto and I'm right narrative".. I'm a pacifist and I'm not interested in going back and forth. No matter how well it's presented and calculated you'll discombobulate the substance, no matter what is presented you will argue because your motive is to just "prove people WRONG" with your type of logic "welp, for your information", clearly as it's very transparent by your response by coming off smug. It's not of importance to prove yourself right Swifto. I could have posted studies but I gave you enough ester eggs to do your OWN leg work as well as the readers..Again, it's an easy find in just a basic search with the results that you provided vs men that are middle age or "aging" that have similar lipid changes due to conditions that impact triglyceride and HDL ratios and other changes that can be associated with the underlining condition, non drug related. The are real facts, not something to "formulate an opinion or interpretation off". These are REAL influences that are indisputable, avoiding them or failing recognizing them is equivalent to refuting there existence.
Answer the questions: Are you saying that the results could not have been impacted by the factors below and you're suggesting I'm pulling it apart by what is logical influential factors that could contribute to the findings with the final results? Let's not get ahead of ourselves, lets stay on topic.. That's what a debate is all about, don't inject other analogies or examples, because they can create distortion.
Could the results have been influenced by the following -
Post #30
This study concludes at the end that "
studies pertaining to androgen substitution are conflicting"... Stated here - androgen substitution therapy on lipids are conflicting but might be favorable..
They faintly suggest they "
might" be "
favorable"..
Maybe I missed it, or didn't see it mentioned, yet environmental circumstances can pose as huge factors with blood readings..
Were these individuals fasted when blood was drawn, did each individual live by the same diets and life habits, was the environment controlled, did the subjects adhere to the same protocols, what about about body fat and mass index and physical activities, how about smoking,alcohol or even history of genetic predispositions? The lack of this information changes the entire dynamics, these adjustments need to be greatly considered..Lack of a controlled diet/lifestyle will dictate results, knowing the regular amount of caloric intake is vital - calories are transformed in to triglycerides and stored as fat, what is the physical characteristics of these men/body mass index? If calories are reduced they will reduce triglycerides especially under physical activities fitness/exercising will lower triglycerides and boost HDL cholesterol, this can drastically shift these numbers .. Furthermore, the data that was presented can be contradicted with the fact that these are "
ageing men", how does this compare to the average age of those that use PED's who most likely are younger or middles aged "supposedly healthy-physically active men" in most cases?
In addition what is the amount of subjects that partaken in these findings? Can the headline of the study or the entire thesis "
Effects of androgen substitution on lipid profile in the adult and aging hypogonadal male".. Explain the outcome and evidence in others other studies that simply contradict the one in question, where subjects weren't even using oral androgens? There's data that supports high triglyceride and HDL ratio can be associated with low test and SHGB levels in middle aged and elderly men.
There's simply to many factors that can contribute here. Pharmacology reports have cited numerous times, throughout the years that mesterolone is practically non-toxic, why would this information be regurgitated by multiple highly credible resources if it was false? In all the reports that I have personally seen, nowhere was there a
controlled study focusing on proving its toxicity and lipid change, in comparison with multiple groups. Yet, we've seen lipid and TG ratio differences with testosterone deficiencies which has been associated with elevated atherogenic lipoproteins, elevated triglycerides, and decreased high-density lipoprotein cholesterol (HDL-C) in males where no exogenous hormones where initially employed - This information is available with basic research, and that's hardly peeling back the layers..
.gif "IP Gear")
