Dementia is multifactorial, in which isn't limited to one biological mediator. Strategically targeting a pathophysiological detriment isn't as simple as popping a couple pills here there.
Although if I was to hyperfixate upon one targeting aspect would be reducing the amount of mitochondrion calcium (Ca2+) influx
Anabolics has the propensity to increase Ca2+.
What's in your APOE (2/3/4) genetic susceptibility, purportedly plays a pivotal role too.
Stewie, are FMRIs better a diagnostic for FTD and Alzheimer’s or will a CT and mri suffice? I know you can see shrinking on CTs but do they do more imaging for a DX?
That’s a good recommendation but I’d Sub alpha-gpc for CDP-choline for the uridine byproduct. And add in idebenone especially. Idebenone being the most potent for brain health and neurotoxicity.