I know I'm kind of beating a dead horse but I'm still wondering if the great pumps/gains from igf-1 lr3 isn't the same thing we'd see with humalin-r or humalog? Has anyone validated actual hyperplasia from it?
I will say this, even if it is working "like insulin", it's a slick insulin and appears to work well for many people. But, does that make it work better than the much cheaper humalog? I really am asking because I seriously don't know. I have used igf-1 lr3 in two different runs and haven't noticed anything that I don't get from humalog personally.
But, I swear I saw site-enhancement from peg-mgf and I suspect that mgf would work even better.
I guess I'm just thinking out loud. I'm open to anyone's insights on this. I'm still a little divided on the whole igf-1/mgf thing. I'd really like to try some des igf-1 with mgf.
There's a lot of info to digest with these drugs and I don't think we have it all figured out yet. For example, some say taking des (1-3) igf-1 with igf-2 lr3 worked wonders. But, why? igf-2 receptors are said to exist mostly in the intestines etc. and not so much in muscle. But, could that be part of why the combo is effective? With the system flooded with igf-2 lr3, is more des making it to "where we want it"? The muscles? Especially considering des' resistance to being broken down and bound to igf binding proteins. Wild speculation, I know. But, this stuff needs to be examined and a good protocol drawn out to get the most out of all of it.