Is Cardarine worth it? Currently on Reta, one injection of 2mg weekly on Wednesday. Microdosing enclomiphine as well at 9.3 mg and taking Cjc no DAC.

[SouthernMuscle]

12/3/25

So I’m 60 years old

11/21/25

I’m in my 50’s.
Just did 3 shows over the last 8 weeks.

6/1/25

I am in my 50's and have not used it in years.

10/1/24

At 53 I’m now thankful for that off time because I still got plenty in the tank

4/10/24

And I’m pretty jacked at 52 years old

Took less than 5min

 

[Sectør]

12/3/25


11/21/25


6/1/25


10/1/24


4/10/24





Took less than 5min

Damn. @MackCoach —why you compulsively lying all the time? You need to get it together man

 

[Virtues]

Guys!! come on. Chill! He obviously has a time machine!

 

[Georgedroyd]

You guys are all too damn funny going at it

 

[Georgedroyd]

You guys are all too damn funny going at it

Also would someone be willing to respond to my previous messages about my diet, training and peptide insight? I’d appreciate it

 

[MackCoach]

In my head I’m about 34 years old.
As they say, age is just a number, you are only as old as you think-feel.

 

[Hyperemia]

In my head I’m about 34 years old.
As they say, age is just a number, you are only as old as you think-feel.

If you're lying about something as stupid as your age, it's safe to assume you're lying about most things. This is why nobody takes you seriously.

 

[bbxtreme]

Hey thank you very much for this response. I’ll let you know today I added high intensity cardio for the first time in a longgg fucking time. It felt fucking great. Was probably the first time in like 6 months since I’ve ran on a treadmill and I felt pretty damn good, did it in intervals for about 16 min total (about 5 min running 2 minute walking pace). Only thing is my heart rate got up to 175 during the peak of the cardio, I’m assuming it’s because my body has never been used to high intensity aerobic before but as I keep doing this every day and body fat goes down it will improve, but let me know if I should keep it in mind. But last time I got my rhr checked it was 58bpm and blood pressure was 89/40 pretty damn low lol. As for diet, the first meal I had was a (white) ready rice packet, 12 grilled chicken nuggets from chic fil a with a fruit cup (I get em free with a school meal pass and figure they’re a good protein option lmk), along with a banana. High potassium foods like bananas and potatoes have never really been a trigger for my episodes I had three bananas today and was chilling lol. Anyways my second well snack was a protein bar I had during a tutoring session. Then after the gym, hit legs, smith squats, leg extensions single standing leg curls and leg curls seated, I ate a bowl of tuna and some dried steak strips. Then about an hour later I had my final meal which was a water based shake, two small bananas, and two scoops of the insane labz brand protein powder with creatine. Let me know what you think man, ima make another post concerning the peptides as well

Not bad for a college kid’s diet (I’m assuming the chicken nuggets are grilled). The whole key to this sport is consistency and planning. Use an app chronometer, etc to track all the macros you are eating daily. You need to find your baseline and go from there.

Same with cardio and your training routine. Log it, track it and stay consistent with what you will stick with. If that’s HIT cardio, cool. If it’s LISS, ok.

There’s no secrets in this game, just consistency of doing the same boring things (ones you enjoy and will stick with) day in and day out over long periods of time.

Spend days, weeks or more on this place. It taught most of us here our foundation, but there’s nothing out there that’s not on this forum for free.

 

[bbxtreme]

In my head I’m about 34 years old.
As they say, age is just a number, you are only as old as you think-feel.

A little humility never hurt anyone man. It’s ok and feels good to acknowledge when we are wrong sometimes. I say that as someone who can be stubborn AF.

 

[pesty4077]

When I want to hear my daily dose of bullshit, I log on here.

 

[pesty4077]

Also would someone be willing to respond to my previous messages about my diet, training and peptide insight? I’d appreciate it

Let me give you best advice you can hear. First, get off all those drugs except the ones that help your condition. You are too young and you don't have time spent in gym. Your physique needs a good diet and sensible approach to training. If you unsure how to eat, instead investing in peptides and other things, invest in a good coach. Someone knowledgeable about your condition.

 

[Massive G]

12/3/25


11/21/25


6/1/25


10/1/24


4/10/24





Took less than 5min

Hilarious. LMAO.

 

[Stewie]

Also would someone be willing to respond to my previous messages about my diet, training and peptide insight? I’d appreciate it

Your situation is exceedingly unique (cautionary) as I alluded to.

I'm vaguely familiar with your specific gene mutation. This is only due to reasons I happen to stumble across literature denoting the unequivocal differentiations on set-of causes associated with periodic paralysis. This was a few year's ago when my daughter's potassium plummeted due to the aforementioned —factaciuos hyperthyroidism (thyrotoxicosis). Your situation— by and-large, in comparison to my daughter's is distinctively different. Although, given the ionic similarities- it's outcome is equally the same. For the interest of digesting newly-found nerdy-stuffage I peeled back the layers of navigating a bit on understanding a bit more on gated channel disorders. I'm far from being an expert— embryotic at best.

Without question, you know your triggers that may disorientate your potassium balance. This would become a very slippery slope if any one person suggested, prescribed a workout, meal planning or peptides– pharmaceutical agents that doesn't know diddly squat about channelopathies.

As discouraging as this may sound, that doesn't mean you can not pursue the endeavors you so seek. Be cognizant about faceless no-name individuals online that purport themselves as a solution to your goals. I can almost assure you that not a single person reading this has the slightest idea on the in's-and-out's on different mediators of ionic influx/efflux. There's deep biochemistry involved.

Here's a suggestion. Look into different molecular structures of compounds (drugs, peptides, those related to lifestyles) that act as a strong candidate's inhibitory motifs Na+/K+ ATPase sodium potassium pump.

 

[Crazy_jay]

Could it be insulin resistance?

Yes. It probably is insulin resistance. Im already late for work...everyday...so I'd like to really expand more on this thread but short and sweet for now.

Potassium shifting causes insulin spikes. Potassium doesnt start shifting back until insulin levels start to drop or become balanced with glucose. Too many shifts, too many insulin spikes...insulin resistance. Welcome to my world. I better not eat myself to insulin resistance either. Bulking is no longer an option for me as I can not afford to have insulin resistance because the shifts never end and my pancreas becomes inflamed. Been dealing with pancreatitis for some time now.

Theres so much to this. Ive been through it all. Ive been to hell and back.

 

[nothuman]

So funny story, I was chatting with a girl who told me she took 20mg cardarine before and got a call from her doctor to say her cholesterol was too low (I asked for specifics but she had no clue).

Then she told me she gave Cardarine to her gay friend and the guy took 3 20mg tabs daily for a while (60mg total!!!). This was a couple years ago and he doesn’t have cancer in case anyone was wondering.

 

[narta]

This was a couple years ago and he doesn’t have cancer in case anyone was wondering.

but but but... the rat studies!

 

[Georgedroyd]

Yes. It probably is insulin resistance. Im already late for work...everyday...so I'd like to really expand more on this thread but short and sweet for now.

Potassium shifting causes insulin spikes. Potassium doesnt start shifting back until insulin levels start to drop or become balanced with glucose. Too many shifts, too many insulin spikes...insulin resistance. Welcome to my world. I better not eat myself to insulin resistance either. Bulking is no longer an option for me as I can not afford to have insulin resistance because the shifts never end and my pancreas becomes inflamed. Been dealing with pancreatitis for some time now.

Theres so much to this. Ive been through it all. Ive been to hell and back.

Yeah I just actually learned a lotttt more about the details and biochemistry of potassium shifts and the insulin spikes that result, which is why in my case with hyper pp, carbohydrates are very beneficial to me because of the insulin spike from them. It’s also why the Retatrutide I take and why GLP1s are being researched now for periodic paralysis because they help level and control insulin spikes and insulin secretion after a meal, which helps the potassium shifts I’m pretty sure. Also now I don’t think that it is insulin resistance and I’ll explain why. When I was taking the CJC 1295 (no DAC, I just took my very last dose last night, I slept like a mf baby, and the dreams are so vivid, I actually can taste things in them real shit). But after I stopped taking the HGH and went on the CJC my fbg was 85 exact, so it dropped by a lot. I think that the adjustment to college life could be causing increases in my cortisol, which would make sense, poor sleep also raises cortisol levels and also increases insulin resistance. But my sleep has been pretty good last couple of weeks. I also learned from my research that things like HGH, injectable IGF-1, insulin and CJC1295 wouldn’t help the hyper pp because of the insulin shifts, which cause more potassium to shift out of the cells. Especially with insulin and IGF1, during exercise potassium is shifting into the cells, and insulin and igf1 causes the opposite to happen so the dramatic and rapid shifts would most certainly cause an episode, so my hypothesis that any kind of insulin manipulation could help was wrong oh well. Retatrutide helps because it stabilizes the levels of insulin secretion and CJC only helped me indirectly because it improved the quality of my sleep by light years, which is my top trigger. GH did as well which is why I don’t experience severe episodes when I used them. But best not to touch them for a while until I can understand the complete biochemistry even more. I still do think it’s a levantes cortisol levels, the declined hair health, acne breakouts would make sense as well, as I’m adjusting to a new setting and routine along with major personal life events over these past couple of months that made me rethink the insulin resistance. But let me know what you think. The variants of our condition are also different but generally the same (we experience many of the same symptoms caused by the same thing in general)

 

[nothuman]

but but but... the rat studies!

I still don’t recommend anyone do that based on one guy I heard about but it was certainly interesting. Apparently he’s the kind of person who will take triple the amount of anything someone tells him to take.

 

[Georgedroyd]

Your situation is exceedingly unique (cautionary) as I alluded to.

I'm vaguely familiar with your specific gene mutation. This is only due to reasons I happen to stumble across literature denoting the unequivocal differentiations on set-of causes associated with periodic paralysis. This was a few year's ago when my daughter's potassium plummeted due to the aforementioned —factaciuos hyperthyroidism (thyrotoxicosis). Your situation— by and-large, in comparison to my daughter's is distinctively different. Although, given the ionic similarities- it's outcome is equally the same. For the interest of digesting newly-found nerdy-stuffage I peeled back the layers of navigating a bit on understanding a bit more on gated channel disorders. I'm far from being an expert— embryotic at best.

Without question, you know your triggers that may disorientate your potassium balance. This would become a very slippery slope if any one person suggested, prescribed a workout, meal planning or peptides– pharmaceutical agents that doesn't know diddly squat about channelopathies.

As discouraging as this may sound, that doesn't mean you can not pursue the endeavors you so seek. Be cognizant about faceless no-name individuals online that purport themselves as a solution to your goals. I can almost assure you that not a single person reading this has the slightest idea on the in's-and-out's on different mediators of ionic influx/efflux. There's deep biochemistry involved.

Here's a suggestion. Look into different molecular structures of compounds (drugs, peptides, those related to lifestyles) that act as a strong candidate's inhibitory motifs Na+/K+ ATPase sodium potassium pump.

I also did some digging on sodium channel blockers, specifically mexiletine, flecainide, and ranolazine. So I learned a lot more about the mutation that causes hyper pp, it’s an SCN4A mutation and the mutation causes a gating pore leak meaning the sodium channels leak inward currents when they shouldn’t. You guys probably know this is just explaining it better for myself. So it seems that ranolazine is best tolerated in individuals, the only more serious side affect I found was possible QT prolongation. What these drugs do is block the abnormal inward flow of sodium caused by the mutation. And the less of the Sodium leak means the membrane is able to be repolarixed and not stay in the depolarized state that would cause paralysis and the muscles will become excitable. Definitely something I’ll keep looking into and when I see my Dr over December break for my periodic paralysis med check in, I will ask him about these compounds.

 

[traininsane11]

. Apparently he’s the kind of person who will take triple the amount of anything someone tells him to take.