Here is a study that speaks of the well-known relationship between Thymus atrophy and sex steroids
To sum up the idea: The thymus is a critical organ that must function properly for the immune system to do its job. The thymus shrinks with age and part of this is due to the sex steroids -as strange as this may sound, testosterone harms the thymus. One way to regrow the thymus is to reduce or completely eliminate your testosterone. All the details you may want are above on the link.
My question is this:
Obviously we do not want to eliminate testosterone (that being sad, if you got something like early stage cancer and want to try this therapy your job a steroid abuser is pretty easy; just stop injecting and your test will plummet and likely not come back for a good while).
Can we take certain AAS that will maintain some or most of the skeletal muscle mass without activating the androgen receptor? If we can, it may be possible to maintain muscle mass to some degree while doing this and during this period the Thymus will regenerate.
Put more simply, which steroid least activates the AR?
To sum up the idea: The thymus is a critical organ that must function properly for the immune system to do its job. The thymus shrinks with age and part of this is due to the sex steroids -as strange as this may sound, testosterone harms the thymus. One way to regrow the thymus is to reduce or completely eliminate your testosterone. All the details you may want are above on the link.
My question is this:
Obviously we do not want to eliminate testosterone (that being sad, if you got something like early stage cancer and want to try this therapy your job a steroid abuser is pretty easy; just stop injecting and your test will plummet and likely not come back for a good while).
Can we take certain AAS that will maintain some or most of the skeletal muscle mass without activating the androgen receptor? If we can, it may be possible to maintain muscle mass to some degree while doing this and during this period the Thymus will regenerate.
Put more simply, which steroid least activates the AR?