Agreed on dopamine agonists.
Side note:
I suggested Mucuna every other day because it has other ligand constituents that metabolize L-Dopa in peripheral tissues, preventing accumulation.
It seems to give a lot of benefits and bypasses sides.
Plus what it does from a GHRH standpoint with peptides is beneficial too.
Interesting stuff:
"The therapeutic effect of Mucuna pruriens has been attributed to a combination of unidentified substances and levodopa and is found to be 2–3 times more effective than compatible doses of levodopa in the animal model of Parkinson’s disease (Hussain and Manyam, 1997). Unlike levodopa, Mucuna pruriens has shown not to induce dyskinesia in the monkey model of Parkinson’s disease (Subramanian et al., 2002) or in patients with Parkinson’s disease (Katzenschlager et al., 2004)."
And:
"Mucuna pruriens cotyledon powder (MPCP) has shown anti-parkinson and neuroprotective effects in animal models of Parkinson’s disease that is superior to synthetic levodopa. In the present study two different doses of MPCP protected both plasmid DNA and genomic DNA against levodopa and divalent copper-induced DNA strand scission and damage. It exhibited chelation of divalent copper ions in a dose-dependent manner. The copper chelating property may be one of the mechanisms by which MPCP exerts its protective effects on DNA."