first, i must to excuse with my bros for my english, but for me is very hard to explain the following theory in english (i'm italian)...
the most important problem with cronic 17aa ingestion is continuous hepatocityes death, liver antioxidants depletion and conseguently liver damage. There are many factor that works together to this damage (too long to explain), but one of the most important is CRONIC ingestion. Why? 17AA AAS cause a strong glutatione depletion in the liver: glutathione acts as a blood substitute in the sulfidryl ligands, binding to itself toxic substances (these would bind to the blood protein without glut.), then carring them to the kidney and pissing them out from the body. Glut. is present in many tissues, but we can find a lot of it in the liver!
But, the presence or the absence of glut. in the liver, is many different when we eat pinkies (i'm taking dbol as example for now, later I must to add some special notes to stanozolol and oxandolone): if U eat dbol when your hepatic glut. levels are low, U make more damage than u would do if you got a glut. filled liver (there isn't any protection). So, what's the best way to keep hepatic glut. in the liver?
this is an example:
week 1 ---> test
week 2 ---> test + dbol
week 3 ---> test
week 4 ---> test + dbol
week 5 ---> test
week 6 ---> test + dbol
week 7 ---> test
week 8 ---> test + dbol
in the "liver relax" days (week 3,5,7) u'll give to your liver, the time it needs to take glut. again and reverse damaged hepatocytes.
In this way, u can use high 17AA dose with no fear. Optional, in the liver relax days, u can use a short acting not 17AA AAS (fina for example). To optimize your liver recovery, i suggest SAMe (powerful antiox. and glutathione production stimulant), injectable glutathione (in italy, we've got "Tad 600"), ALA (we already know this boy), NAC (to enhance glut. substrates), vit C.
Note: usually, 17AA AAS cause a fall of liver glutathione levels, while stanozolol and oxandrolone don't. Oxandolone isn't liver toxic because it hasn't got any metabolities (toxic or not). In other words, when u eat var, U piss var, when U shot test, U piss 17-cheto-steroids... do U know what I mean?
stanozolol doesn't cause a fall in hepatic glut. levels; this mean that this cycle planning could be more effective for liver recovery if U ingest 17AA as dbol, abomb, metiltest, mesanolon, and other... (glut. depletors)
I hope U understood my idea. Now i'm curious to know what do U think about...
the most important problem with cronic 17aa ingestion is continuous hepatocityes death, liver antioxidants depletion and conseguently liver damage. There are many factor that works together to this damage (too long to explain), but one of the most important is CRONIC ingestion. Why? 17AA AAS cause a strong glutatione depletion in the liver: glutathione acts as a blood substitute in the sulfidryl ligands, binding to itself toxic substances (these would bind to the blood protein without glut.), then carring them to the kidney and pissing them out from the body. Glut. is present in many tissues, but we can find a lot of it in the liver!
But, the presence or the absence of glut. in the liver, is many different when we eat pinkies (i'm taking dbol as example for now, later I must to add some special notes to stanozolol and oxandolone): if U eat dbol when your hepatic glut. levels are low, U make more damage than u would do if you got a glut. filled liver (there isn't any protection). So, what's the best way to keep hepatic glut. in the liver?
this is an example:
week 1 ---> test
week 2 ---> test + dbol
week 3 ---> test
week 4 ---> test + dbol
week 5 ---> test
week 6 ---> test + dbol
week 7 ---> test
week 8 ---> test + dbol
in the "liver relax" days (week 3,5,7) u'll give to your liver, the time it needs to take glut. again and reverse damaged hepatocytes.
In this way, u can use high 17AA dose with no fear. Optional, in the liver relax days, u can use a short acting not 17AA AAS (fina for example). To optimize your liver recovery, i suggest SAMe (powerful antiox. and glutathione production stimulant), injectable glutathione (in italy, we've got "Tad 600"), ALA (we already know this boy), NAC (to enhance glut. substrates), vit C.
Note: usually, 17AA AAS cause a fall of liver glutathione levels, while stanozolol and oxandrolone don't. Oxandolone isn't liver toxic because it hasn't got any metabolities (toxic or not). In other words, when u eat var, U piss var, when U shot test, U piss 17-cheto-steroids... do U know what I mean?
stanozolol doesn't cause a fall in hepatic glut. levels; this mean that this cycle planning could be more effective for liver recovery if U ingest 17AA as dbol, abomb, metiltest, mesanolon, and other... (glut. depletors)
I hope U understood my idea. Now i'm curious to know what do U think about...