I did some digging after reading the russian study and ran across this where glycerol neutralizes the acids in the blood. Technically speaking, stopping free radical peroxidation also makes glycerol an anti-oxidant.
'- Glycerol also binds with and neutralizes the free fatty
acids that are the primary cause of the aging process that
results form free radical peroxidation -- particularly the
abnormal fatty acids with conjugated double bonds which
most accelerate the aging process.'
Finally some 'redemption' for glycerol and an explanation as how it works!
Previously the worry with glycerol was that by its action as we know, it increases blood volume, this phenomenon by logic dictates that an increase in volume therefore dilutes hemoglobin and hematocrit and if that happens how can performance be improved?
I didn't know and couldn't explain it, but finally it has come out that volume changes to hematocrit and hemoglobin have NO EFFECT ON VO2!
But the kicker is that plasma expansion BOOSTS VO2 MAX!
Therefore, your normal load will be the same and your vo2 max will go up and glycerol is THE ONLY 'normal' CHEM YOU CAN TAKE TO DO THAT! And it's the ONLY way I know of to do it!
This first study is in pdf so you have to go there and there is another which I'm trying to find, but the link elf sent me doesn't work:
http://jap.physiology.org/cgi/reprint/101/3/707.pdf
this is the second one which I can't pull:
BLOOD VOLUME, NOT HEMOGLOBIN-CONCENTRATION IS RELATED TO VO2... : Medicine & Science in Sports & Exercise
Then I found a third and it discusses blood volume AND AGING and how a sedentary life DECREASES BV which seems to be protective!
Validity of V?o 2?maxin predicting blood volume: implications for the effect of fitness on aging | Regulatory, Integrative and Comparative Physiology
Quote:
Our findings have particular implications for the effect of physical activity and fitness on aging. Our data support the notion that BV actually increases with age but that this relationship may be masked by a sedentary "Western" lifestyle that can often accompany the aging process. This notion is further supported by the observations that BV can be increased with regular physical activity in elderly individuals to the same relative degree compared with younger people (5) and that reduction in BV associated with aging in sedentary subjects was removed in physically active subjects (21). The contraction of BV may be associated with adverse impacts on risk factors for cardiovascular disease such as elevated low-density lipoprotein cholesterol, increased whole blood viscosity, and stimulation of sympathetic nervous activity (33). In turn, sympathetic hyperactivity is typically reported in patients with essential hypertension and chronic renal failure and is associated with poor prognosis and increased risk of sudden death (23). The relationships between BV, sympathetic activity, and progressive cardiovascular disease may reflect a protective nature of increased BV against development of coronary heart disease (CHD) with aging. If it is true that BV increases with age when a sedentary lifestyle has been removed (21), then perhaps a sedentary lifestyle is actually acting to remove a natural CHD protective factor. For example, if less viscous circulating blood or sympathetic activity results from increasing BV with regular physical activity during the aging process, then various risk factors associated with CHD such as platelet aggregation, arterial thrombosis, or cardiac arrhythmias are less likely to occur. The literature supports the observation that, in the Western world, BV does decrease with age (6, 11, 15, 21, 30). However, our data provide compelling evidence that reduced BV with age may be a result of a sedentary, high-caloric lifestyle rather than the aging process. Therefore, perhaps one of many important benefits of maintaining physical activity and fitness during aging is the resultant expansion of plasma and BV that provides a protective effect against development of cardiovascular disease.