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pancreatitis and bbuilding? AAS

TIMMO

New member
Registered
Joined
Oct 8, 2005
Messages
144
hey everyone, ive just came out of hospital with an enlarged pancreas and being god dam ill.
now, who on this board has had pancreatitis and did they find a cause? because they didn't with mine.
ive had full blown pancreatitis 3 times which resulted in me being in intensive care for 2 weeks each time. and loosing 40lbs of body weight each time.
ive had edoscopys, colonoscopys, ultrasound scans and full blood work every 2 months and still call it idiosyncratic pancreatitis( cant find a cause).
didn't drink much alcohol before the first time and certainly not sinse, haven't used any aas for 4+ years and im on nebido test inj from my endo every 6 weeks(I live in the uk ,this is all we have and its free).
please please, anyone that has had pancreatitis, did they find a cause or just blame the aas you haven't used for years?
does any one have advice on specific food intake or anything that may help?
before this time in hospital I was 5'101/2 and 250ishpounds on trt only, ive been bb for 23years now so I do know a thing or two but this pancreas trouble has me completely stumped? and is ruining any gym work, exercise and my life.

thanks for any help/advice in advance TIMMO
 
hey everyone, ive just came out of hospital with an enlarged pancreas and being god dam ill.
now, who on this board has had pancreatitis and did they find a cause? because they didn't with mine.
ive had full blown pancreatitis 3 times which resulted in me being in intensive care for 2 weeks each time. and loosing 40lbs of body weight each time.
ive had edoscopys, colonoscopys, ultrasound scans and full blood work every 2 months and still call it idiosyncratic pancreatitis( cant find a cause).
didn't drink much alcohol before the first time and certainly not sinse, haven't used any aas for 4+ years and im on nebido test inj from my endo every 6 weeks(I live in the uk ,this is all we have and its free).
please please, anyone that has had pancreatitis, did they find a cause or just blame the aas you haven't used for years?
does any one have advice on specific food intake or anything that may help?
before this time in hospital I was 5'101/2 and 250ishpounds on trt only, ive been bb for 23years now so I do know a thing or two but this pancreas trouble has me completely stumped? and is ruining any gym work, exercise and my life.

thanks for any help/advice in advance TIMMO

Do you have Acute or Chronic pancreatitis?
 
i was hospitalized for pancreatitis once...
I was on low a dose.1cc a week tornel t200(notoriously underdosed).
but I think it was NO2(was a new thing at the time).
I had gotten sick from it before but I was using torbugesic(strong nubaine for horses),so I just shot up(many,many times)and rode it out.
the second time(when I was hospitalized)I wasn't using painkillers.
same test dose and NO2...
been along time...at least eight years and no issues.
common sense tells me it was the NO2.
probably wont help but that's what happened to me.
I know a guy with chronic pancreatitis but hes an alcoholic and wont stop drinking.
 
thanks for the replys.
Emeric, ive had 3 cases of acute pancreatitis and ended up in intensive care, and nearly died with amylase at over 15000, the gastroenterologist said he'd never seen them that high ,ever.
at the moment ive got an enlarged pancreas(ultrasound scan) amylase only 160, but im slowly starting to feel better,and my pancreas seems to be working properly.
this is 41/2 years sinse the last acute attack and its really fucking my life up now.
don't drink alcohol or any gear, just nebido trt every 6 weeks from endo.
please ,what can I do to help myself?

thanks TIMMO
 
did a doctor give you a list of foods to avoid???
might be diet related.probably diet related.
maybe start a food journal...then you can look back and see if there is a pattern to certain foods and flare ups.
feel for ya man,pancreatitis is really awful.
 
way2tense,
I did have some fried food the day before this problem, I hadn't had a fry up for 5-6 months so could well be diet related.
I eat healthy all year normally but its nice occasionally to have a junk meal? I did think a fatty diet was related to gaulstones but I could be wrong?
I lost 10lbs in 4 days in hospital and cant see me going back to the gym for a few more weeks, so this sucks big time
 
Had it several months back, in hospital and lost 25lbs in 4days...complete misery. I had acute, havent drank in many many years and on TRT is all. No cause or other issues found. I stay away from fatty foods and generally cleaner diet...no problems since (knock on wood). was told approx. 1/4 of cases have no found cause.
 
Did they check for any biliary obstruction, like Gallstones or anything?

Here is the complete run-down of Pancreatitis, copied from the nutrition care manual. You have to be a member of the ADA to view it, so i'll copy and paste it here so that it can be read.

******************************************************
Overview of Pancreatitis

Pancreatitis is a complex condition involving an inflammation of the pancreas. The condition can be both acute and chronic; can range from mild to severe; and, in the case of chronic pancreatitis, can take several years to evolve.

Acute pancreatitis is most often associated with alcoholism and biliary tract obstruction (Nagar, 2004). Pancreatitis may evolve from other medical conditions such as cystic fibrosis, hypertriglyceridemia, hypercalcemia, renal failure, or infectious causes such as hepatitis or mumps. Some medications such as diuretics (furosemide) or antibiotics (tetracycline); trauma; surgery; or other tests can lead to acute pancreatitis. Still, alcohol abuse accounts for 70% to 80% of all cases.

Risk factors for acute pancreatitis include the following:

Previous gallbladder disease
Chronic alcohol ingestion
Obesity
Exposure to certain toxins or drugs
Cystic fibrosis, hereditary conditions, and chemical exposure can potentially lead to chronic pancreatitis.


Background Information
The pancreas, a gland lying posterior to the stomach and alongside the duodenum, performs both exocrine and endocrine functions.

Exocrine functions include the secretion of digestive enzymes, bicarbonate, and electrolytes. The pancreas may secrete up to 2.5 L secretions each day. The release of secretions are controlled by a host of neurohumoral factors, including the hormones gastrin and secretin. Neuropeptides involved in pancreatic function include somatostatin, vasopressin (vasoactive intestinal polypeptide), and gastrin-releasing peptide (bombesin).

Endocrine functions center on the secretion of insulin and glucagon, which are pivotal in the metabolism of glucose, fatty acids, and amino acids. When these normal processes of digestion and absorption are interrupted, the resulting signs and symptoms of acute pancreatitis occur. Pancreatitis can either be acute or chronic.

Chronic alcohol abuse is a leading contributing factor in acute and chronic pancreatitis, but these conditions differ from one another in several important ways:

Acute: The acute inflammation that occurs is generally associated with acute abdominal pain; nausea and vomiting; elevated serum levels of pancreatic enzymes; and C-reactive protein, and can be accompanied by infection. Except in very severe cases, the pancreatic function usually returns to normal after recovery from the acute episode.

Chronic: Chronic pancreatitis may be asymptomatic or may be characterized by intermittent, recurrent abdominal pain; serum levels of pancreatic enzymes are often normal. Chronic pancreatitis leads to permanent tissue damage with resulting permanent pancreatic insufficiency.


Disease Process
The exact mechanisms that lead to pancreatic injury are not fully understood. However, a common characteristic seems to be premature activation of trypsin within the pancreas, resulting in autodigestion of the pancreatic cells. The enzymes released by destroyed pancreatic cells eventually reach the bloodstream, causing elevated serum amylase and lipase levels. Systemic complications can include the following:

Shock
Respiratory failure
Sepsis


Signs and symptoms are as follows:

Abdominal pain radiating to the back
Nausea
Vomiting
Steatorrhea


Biochemical indices for diagnosis of pancreatitis incude the following:

Serum lipase
Serum amylase
Secretin stimulation test
Glucose tolerance test
Ranson's criteria (1977), computed tomography severity index, or Apache scores are used to grade the severity of the disease and can be additionally confirmed using the following procedures:
****Endoscopic retrograde cholangiopancreatography
****Computed tomography
****Ultrasound

Chronic pancreatitis is the result of irreversible damage to the pancreas that is caused by repeated inflammation and results in destruction of the exocrine and eventually the endocrine tissue.

The primary intervention for chronic pancreatitis is to minimize symptoms of pain and malabsorption by avoiding exacerbating factors (alcohol and high fat intake) and using pancreatic enzyme replacement as needed. In addition, endocrine abnormalities such as diabetes mellitus will need to be addressed.

For more information, see Pancreatitis–American Gastroenterological Association (accessed July 15, 2010).


Biochemical and Nutrient Issue
The primary intervention for acute pancreatitis is to prevent stimulation of the pancreas and thus reduce the release of its secretions. Thus, if the patient does not receive nutrition support, there will be risk for inadequate energy, protein, vitamins, and minerals.

In chronic pancreatitis, the symptoms of malabsorption and endocrine complications may lead to maldigestion and malabsorption of nutrients. Avoidance of food to decrease pain may result in malnutrition.


Implementation of the Nutrition Intervention - Oral Intake
Patients who undergo prolonged nutrition support or NPO (nil per os, or nothing by mouth) status may develop a fear of eating or aversions to specific foods that they associate an exacerbation of symptoms.
Feeding issues may present in the opposite manner—that is, patients may have a desire to eat when it is imperative that they maintain NPO status for recovery.

Close observation and counseling is necessary in both of these circumstances, and patients should be encouraged to discuss their issues with the health care team so that an appropriate plan of care can be determined and implemented.

As the patient's amylase and lipase begin to trend downwards, oral intake may be initiated. The historical progression from NPO to oral intake has included transition from clear liquids to a low fat solid food diet. Monitoring for any gastrointestinal complaints will alert the practitioner for any intolerance to the oral diet.


Biochemical Data, Medical Tests and Procedures

To diagnose pancreatitis, perform the following laboratory tests:
White blood cell count
Serum glucose
Serum lipase
Amylase
Lactic dehydrogenase (LDH)
Aspartate aminotransferase (AAST)


The degree of inflammation (and thus the severity of the disease) may be assessed by the following:
C-reactive protein
Evaluation using Ranson’s criteria, Apache score or Computed Tomography Severity Index


It is also common practice to use a combination of criteria that distinguish the severity of the disease. Common criteria include Ranson’s criteria, Apache score, and the Computed Tomography Severity Index. Ranson’s criteria (1977) are as follows:

Admission:
Age >55 years
White blood cell count >16,000/mm3
Blood glucose >200 mg/dL (especialy in nondiabetic patients)
LDH >350 IU/L
AAST >250

After initial 48-hour period:
Decrease in hematocrit by ≥10%
Increase in serum blood urea nitrogen by >5 mg/dL
pO2 of <60 mm Hg
Base deficit >4 mEq/L
Fluid sequestration >6 L


To follow hydration and acid-base status, monitor the following:
Serum electrolytes
Arterial blood gases


Other biochemical abnormalities may include the following:
Hypertriglyceridemia
Hypocalcemia (Rettally, 2003)


To determine presence of pseudocysts or necrosis, other diagnostic tests may include the following:
Computed tomography
Ultrasound
Endoscopic retrograde cholangiopancreatography


Laboratory Value Norms

Laboratory Normal Range: Adult Values

Amylase 25-125 U/L
Lipase 0-417 U/L
Lactate dehydrogenase 313-618 U/L
Serum glutamic-pyruvic transaminase 10-60 U/L
Serum glutamic-oxaloacetic transaminase 5-40 U/L
C-reactive protein 0
Hemoglobin 12-16 g/dL, women; 13.5-17.5 g/dL, men
Hematocrit 37% to 47% (women); 40% to 54% (men)
Glucose 70-110 mg/dL
Blood urea nitrogen 8-26 mg/dL
Creatinine 0.6-1.3 mg/dL
Sodium 135-155 mmol/L
Potassium 3.5-5.5 mmol/L
Phosphorous 2.5-4.5 mmol/L
Chloride 98-108 mmol/L
Calcium 8.7-10.2 mg/dL
Carbon dioxide 24-30 mmol/L
Osmolality 275-295 mOsm/kg H20
Vitamin D (25-hydroxy) 16-74 ng/dL

Nutrients Below Target Due to Nutrition Prescription

Carbohydrate, protein, and fat should be balanced according to standard guidelines when nutrition support is provided unless hyperglycemia, hypertriglyceridemia, or other metabolic complications necessitate substrate modification. Special attention to fat-soluble vitamin status may be needed when steatorrhea and other signs of malabsorption are present.

Dietary fat intake may be lower as a result of steatorrhea and malabsorption.

Standard multivitamin/mineral therapy should be considered during the recovery phase until normal eating habits have been well established and signs and symptoms of deficiency are not present.

Micronutrient deficiencies common in chronic alchoholism include thiamin; pyridoxine; folate; vitamins C, A, and K; zinc; and magnesium (Sucher, 2011 in press).
 
thanks guys, that's the sort of info im looking for , so a low fat diet is of importance now?
thanks BMJ ill show this to my doctor and see what they say or do?
ive not got gaulstones and ive had an ultrasound scan this time around, my liver is smooth contour with no focal leisions
CBD is not dilated
the spleen is normal
both kidneys are normal in size shape and echogenicity.
amylase now 130
WCC 11.8
CRP 4
a bulky pancreas but no other significant finding

again a big thanks for your input

do we have any doctors on this board I could ask to look at this thread and hopefully some input?
 
My doctor warned me if I didn't bring my triglycerides
down, I could come down with pancreatitis .
 
thanks for the replys.
Emeric, ive had 3 cases of acute pancreatitis and ended up in intensive care, and nearly died with amylase at over 15000, the gastroenterologist said he'd never seen them that high ,ever.
at the moment ive got an enlarged pancreas(ultrasound scan) amylase only 160, but im slowly starting to feel better,and my pancreas seems to be working properly.
this is 41/2 years sinse the last acute attack and its really fucking my life up now.
don't drink alcohol or any gear, just nebido trt every 6 weeks from endo.
please ,what can I do to help myself?

thanks TIMMO

PM sent, call me.
 

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