DISCUSSION:
Now, on to what the consensus on the literature has been----
Peter Bond wrote a classical report on the metabolism of Primo in skeletal muscle and the mechanism behind its reduced anabolism, as well as the weaknesses of the Hershberger Assay: https://peterbond(dot)org/post/methenolone-primobolan-3a-reduction
Building upon that foundation, here is a graph from a 1965 study [1] which analyzed various AAS under strictly controlled conditions-- specifically Primo, Deca, Dbol, Anadur (nandrolone hexylphenylpropionate, a 21-day half life form of nandrolone), and Androxan (oral similar to androstanozole). It is a
very strong study designthat studied the human effects of the compared AAS under precisely manipulable conditions (»). That is, patients were tube-fed, immobilized, and the researchers could tease out the anabolic effects of the AAS studied with dietary manipulation (protein and caloric intake were manipulated) precision:
In one patient (Case 1) a 100mg bolus of Primo was administered resulting in an uncomplicated 16 day positive nitrogen balance with a total nitrogen retention of 30.8g [1] on a caloric surplus (55%>BMR) and protein intake of 1.65 g/kg b.w. See Figure 1(N) [above].
This basically matched the nitrogen retention of a woman on an equivalent caloric and protein diet (45%>BMR), 1.5 g/kg b.w (30.6 g, 18 day positive nitrogen balance) [1].
The following Table shows that Primo at 100mg increased lean tissue by about 1/2 the amount as 50mg (!) of Deca:
(Written by
@Type-IIx )
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www.ncbi.nlm.nih.gov
)
