well, i have ran 2mg injectable methyltrienolone pre workout for 3 weeks and my bloodwork had ALT and AST somewhere around 70-80...
ive read about people getting jaundice and severe liver damage from 1mg methyltrienolone for 2 weeks taken ORALLY:..
am i just very resilient then?
i rather definitely think liver damage is GREATLY reduced with the injectable form.
my hypothesis is:
Bost damage to the liver is done because the whole amount of the orally taken methylated steroid hit the liver AT ONCE after ingestion, then slowly circulates through the bloodstream.
with injection, it takes much longer for 100% to reach the bloodstream PLUS there is no initial hit when the steroid is "digested".
this makes sense to me and also goes hand in hand with how i feel when on "injectable orals" vs "oral orals"
Methylated drugs delivered via injection reach the bloodstream very quickly. Remember, they aren't esterfied....and neither are they suspended in crystal form, so there is nothing to hinder absorption.
Secondly, your theory that methyls are less toxic when injected because they are processed more slowy (lacking the "hit") does not hold up under scrutiny. For one, most people, when using injectables, will only inject once daily--meaning the entire daily dose is absorbed over just a few hours. However, when taking methyls orally, most people administer them according to half-life, so smaller amounts of the drug are being delivered into the sytem over the course of the day.
Besides, the half-life will expire at the same rate regardless of administratioin route. This means the entire dose will be metabolized by the liver in full each and every day, regardless of delivery method.
Basically, neither of the above-mentioned factors have a significant effect on liver toxicity.
Remember, about 95% of mnethylated AAS are metabolized by the liver anyway. It's not like the liver is "spared" just because the steroid avoids first pass...the drug just takes a bit longer to be fully metabolized, which all happens in the liver regardless.
The reason your liver enzymes weren't that high is because M-tren is not nearly as toxic as originally thought. All of those posts about how M-tren will cause liver destruction in just a few weeks were not based on fact or any type of real-world experience. The reality is that using M-tren, whether oraly or via injection, is not excessivley toxic when the dosage is kept reasonable and the cycle duration short.
I have seen many labs of people using both forms of delivery and there has been very little difference in terms of toxicity. Total dosage and length of administartion are much more important factors. Like I said previously, injecting methyls doesn't make a big difference in terms of toxicity..and I have seen hundreds of labs to comnvince me.
It would be awesome if we
"greatly" reduce a methyl's tocivity by injecting it, which you claim happens, but it just doesn't work that way.
I will look for some studies on PubMed demonstrating the toxicity profile of both injectable and oral winstrol. I read them years ago, but need to do some searching.