The bold is bs and the rest is your opinion... Not the facts.
A major side effect of growth hormone (GH) administration is fluid retention.
Growth hormone and fluid retention. [Horm Res. 1999] - PubMed - NCBI
However, the effect of growth hormone was mostly due to fluid retention, the researchers found.
**broken link removed**
It is concluded that short-term administration of GH to short normal children results in a transitory mild retention of sodium and a secondary water retention, and suggests that the primary event leading to sodium retention during the early phase of GH therapy of short normal children is an inappropriate increase in PRA.
Water and sodium retention during short-term admini... [Horm Res. 1998] - PubMed - NCBI
Look, the bottom line is that Growth Hormone administration leads to increased
aldosterone . Particularly excessive levels are even more prone to cause
this.
I'm open to reading about the facts that you've gathered on this subject. I'm not emotionally invested in this, so please fill me in on what you've discovered. Until then it might be useful to consider this phenomenon, if not a fact, then a scientifically scrutinized and verifiable consequence of Growth Hormone usage, especially in the dosages we're using them.
To the OP, were you using anything else when you tried the domestic version?
Go out and buy some Ester-C. It has done amazing things for the edema I was experiencing. Don't bother with Caber, as this isn't a prolactin issue, even though excessive GH does bind to prolactin receptors. It's that GH binds to receptors in the kidneys and tells them to hold on to sodium. The best thing to combat that side effect is through Angiotensin Converting Enzyme inhibitors, like captopril. That and make damn sure you're eating twice as much potassium rich foods as you are sodium rich foods. That's just plain good advice anyways.
As for why some of the GH floating around is causing excess retention and others is causing almost no retention? I can only guess because I don't know the science behind the production of this stuff. But GH does cause sodium retention, there is little reason for me to doubt this, therefore the idea that more pure GH causes less retention just doesn't jive with me. It could be that impure GH has little proteins left in it that bind to the renin receptors in the kidneys but are not complete aminos so they don't bind to the GH receptor, but that's still a stretch considering I'm out of my league on the biology of this.
But like I said, I'm far from attached to my findings, so anything else people can share with me that has been peer reviewed and has multiple findings to back it up - I'm all ears.
