I'm not too worried about this causing cancer as the doses look very high and drawn out compared to what humans would research.
However, I would be worried about spending $100+ on a month's supply of something...what types of results are board members seeing from this? Obviously an increase in endurance and an improved blood lipid profile is great, but what about results as far as fat loss/muscle gain are we talking about?
Sorry I got busy at work. Because of AA use and its subsequent damage on lipid profile in the body, true GW501516, should be considered. Especially when AA involves the use of Stanozolol or boldenone both of which to an extend create CD through lipid profile or increase levels of Erythropoiesis. The increase in HDL has shown to retard CD issues if not lower the risk of CD.
Considering other weight loss alternatives and the much cheaper prices, I'm wondering what all the fuss is about. DNP, T3 won't negatively impact your lipid profile if not used foolishly...a good diet/cardio regiment with some quality supplements can help maintain a good lipid profile and increase endurance...plenty of other peptides/aas that can help increase size & strength and endurance.
I cannot speak for DNP. But Thryroid hormones can have an important effect on lipid profile which is why screening for thyroid dysfunction is critical in all dyslipidemic patients, as well as in all patients with unexpected improvement or worsening of their lipids.
T3 will negatively impact your lipid profile by controlling the sterol regulatory element-binding protein-2, which regulates LDL receptor’s gene expression. T3 has also been associated with protecting LDL from oxidation.
Another effect of T3 is the increased levels of ApoAV which have been associated with decreased levels of TGs
Decrease in HDL-C levels is also observed in high levels of T3, due to increased CETP-mediated transfer of cholesteryl esters from HDL to VLDL and increased HL-mediated catabolism of HDL.
And though I do believe in omega's 3s remember that everyone is different. For example omega-3 (often consumed as ALA, which the body converts to EPA) further breaks into eicosanoids, of which interest here are the prostaglandins that inhibit blood clotting. As a result, EPA is linked to heart health (no blood clots) and is also linked to decreased LDL, increased HDL, decreased TG, and decreased inflammation. However omega-s should not be given in individuals with bleeding disorders, taking anticoagulation meds, about to have surgery or have been diagnosed with uncontrolled HTN as the risk of hemorrhagic stroke increases.
Still the level of dislipidemia that GW modulators achieve vs others is higher (clinically and not anecdotal).
CARDIO AAS and lipid profile.
Because of endogenous use of AA, your body that would normally break cholesterol into sterols and hormones, in particular testosterone will not do so at the same rate. Level hepatoxicity as well as blood thickening will alter lipid profile (HDL low, LDL high, trig +/- as it has more to do with genes and diet). The positive effect of aerobic exercise in lipid levels will not match pound per pound the physiological restrains on lipid, I am not saying you won't be healthy. I work with mice and sometimes bigger mice so take my input as a grain of salt.
By far the only people I have seen to maintain good lipid profile white on AAS were wrestlers (real wrestlers) since their aerobic training and maximal strength output were done for 4+ hours a session.
DO you need GW-501516 to maintain basal lipid profile while on AAS or any other hormone (t3, gh etc). For some no, for some yes.
I never said you needed to, but cost-to-benefit-ratio I think is a good option to make sure your CV risks are minimized as much as possible.
Effects:
(1) inc. hdl
(2) dec ldl
(3) dec tryglicerides
(4) Minimal liver repair through increase in liver fibrosis and stellate cell proliferation.
(5) increase lipid usage (combustion) and (6) antidiabetic action; for these two you will see sharp positive results the fatter you are and minimal results the leaner you are because it is based on the glucose-fatty-acid system.
Hope this helps
increases lipid combustion and exerts antidiabetic action