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High test vs Low Test with other anabolics/androgens?

Do you like high or low test?

  • I run high test with lower anabolics/androgens

    Votes: 42 26.1%
  • I run low test with higher anabolics/androgens

    Votes: 87 54.0%
  • I just blast high on everything :/

    Votes: 32 19.9%

  • Total voters
    161
Can you expand on this?
I have tried this as part of my trt from reading Dr.G's posts and I'm not sure if its the right thing right now. I can't sleep even on that low dose! The main reason for me was for the nutrient partitioning effects as I have digestive issues.

x2....is it retarded for a cruise and trt alike...or just a cruise?
 
Retarded. Cruise is for recovery, tren is the last thing you want. If you aren't dieting for a contest, tren isn't worth it. I won't even use for contest prep anymore.



I have used all the AIs, pharm grade, after years on aromasin, I went back to arimidex and am staying with arimidex. Aromasin has nasty digestive side effects, and I don't believe it is any easier on lipids. 200 to 600 test I use .25mg/day.



Way too much. BTW I have done bloodwork with both letro and aromasin on 1g test, they are the same strength.

so both health/effects wise you feel adex is the better option than aromasin? it seems asin gets all the love as the easiest on lipids and all, curious to hear your thoughts
 
Can you expand on this?
I have tried this as part of my trt from reading Dr.G's posts and I'm not sure if its the right thing right now. I can't sleep even on that low dose! The main reason for me was for the nutrient partitioning effects as I have digestive issues.

Isn't the point of a cruise to give your body a break? I read most of Dr. G's posts, I disagree with almost everything he says.
 
What are your thoughts on 100 test 100 tren as HRT ?

I am on the 100test 100 tren. I believe the tren is a little added bonus to help ya keep your gains while cruising. I like it.
 
I am on the 100test 100 tren. I believe the tren is a little added bonus to help ya keep your gains while cruising. I like it.

have you done a blood work recently ?
 
Isn't the point of a cruise to give your body a break? I read most of Dr. G's posts, I disagree with almost everything he says.

But with all due respect, hes a very accomplished Doctor and you are a really intelligent "bro".
 
But is aromasin at 25mg twice daily with fats enough to suppress e2 on heavy cycles including 1g+ test and dbol?

That is way too much and I'd question if it is legit aromasin.
 
Way too much. BTW I have done bloodwork with both letro and aromasin on 1g test, they are the same strength.

Please get very specific as I think this is interesting, to say the least. Do you just mean the same strength in terms of e2 suppression? What dosages are the same strength?
 
Last edited:
But with all due respect, hes a very accomplished Doctor and you are a really intelligent "bro".

Most successful doctors don't know shit about endocrinology or biochemistry (or even nutrition for that matter). It is a necessary side effect of having a very broad range of knowledge, you could also accuse the specialists of having too narrow of a range to diagnose a patient properly.

Please get very specific as I think this is interesting, to say the least. Do you just mean the same strength in terms of e2 suppression? What dosages are the same strength?

I have posted on this extensively, including research. In males, the standard daily dose of all three 3rd gen AIs is almost exactly the same. Beyond that I have done some of my own research on myself and also found the same exact result.

Letro at standard daily dose (2.5mg) reducing E2 by 50% in males.
Letrozole once a week normalizes serum testosterone in obesity-related male hypogonadism

Arimidex at the standard daily dose (1mg) reducing E2 by 50% in males.
**broken link removed**

Aromasin at the standard daily dose (25mg) reducing E2 by 62% after 12 hours in males. 38% after 24 hours (average of 50%).
**broken link removed**

My own results comparing Letro with Aromasin (Aromasin at 20mg not 25mg unfortunately).
http://www.professionalmuscle.com/forums/beginners-forum/65807-estradiol-1g-test.html

I could explain the likely reason why they are weaker in males, why they all work about the same, and why you cannot block higher amounts even with much higher doses, but that part would be pure broscience (although I would call it a hypothesis based on knowledge of biochemistry). I know some of you are aware of my theory.
 
Last edited:
Most successful doctors don't know shit about endocrinology or biochemistry (or even nutrition for that matter). It is a necessary side effect of having a very broad range of knowledge, you could also accuse the specialists of having too narrow of a range to diagnose a patient properly.

Pretty sure his resume and credentials in the field i.e. at Schering make him a bit different than "most" doctors. But that's just my opinion..
 
Most successful doctors don't know shit about endocrinology or biochemistry (or even nutrition for that matter). It is a necessary side effect of having a very broad range of knowledge, you could also accuse the specialists of having too narrow of a range to diagnose a patient properly.



I have posted on this extensively, including research. In males, the standard daily dose of all three 3rd gen AIs is almost exactly the same. Beyond that I have done some of my own research on myself and also found the same exact result.

Letro at standard daily dose (2.5mg) reducing E2 by 50% in males.
Letrozole once a week normalizes serum testosterone in obesity-related male hypogonadism

Arimidex at the standard daily dose (1mg) reducing E2 by 50% in males.
**broken link removed**

Aromasin at the standard daily dose (25mg) reducing E2 by 62% after 12 hours in males. 38% after 24 hours (average of 50%).
**broken link removed**

My own results comparing Letro with Aromasin (Aromasin at 20mg not 25mg unfortunately).
http://www.professionalmuscle.com/forums/beginners-forum/65807-estradiol-1g-test.html

I could explain the likely reason why they are weaker in males, why they all work about the same, and why you cannot block higher amounts even with much higher doses, but that part would be pure broscience (although I would call it a hypothesis based on knowledge of biochemistry). I know some of you are aware of my theory.

What always threw me off about your tests Kaladryn is the presence of tren hex in your cycle. How do those effect your e2 values and what are your true e2 values if the test is flawed cause of the tren. Are you actually somewhere near 20-40 e2 and your hex is inflating the values 75+ for example?
 
Good point pieguy. I hadn't realized that was a component of his cycles. How much Hex was being run?


As a side note, Id like to know all about your experiences and opinions with tren hex Kaladryn.
 
Most successful doctors don't know shit about endocrinology or biochemistry (or even nutrition for that matter). It is a necessary side effect of having a very broad range of knowledge, you could also accuse the specialists of having too narrow of a range to diagnose a patient properly.



I have posted on this extensively, including research. In males, the standard daily dose of all three 3rd gen AIs is almost exactly the same. Beyond that I have done some of my own research on myself and also found the same exact result.

Letro at standard daily dose (2.5mg) reducing E2 by 50% in males.
Letrozole once a week normalizes serum testosterone in obesity-related male hypogonadism

Arimidex at the standard daily dose (1mg) reducing E2 by 50% in males.
**broken link removed**

Aromasin at the standard daily dose (25mg) reducing E2 by 62% after 12 hours in males. 38% after 24 hours (average of 50%).
**broken link removed**

My own results comparing Letro with Aromasin (Aromasin at 20mg not 25mg unfortunately).
http://www.professionalmuscle.com/forums/beginners-forum/65807-estradiol-1g-test.html

I could explain the likely reason why they are weaker in males, why they all work about the same, and why you cannot block higher amounts even with much higher doses, but that part would be pure broscience (although I would call it a hypothesis based on knowledge of biochemistry). I know some of you are aware of my theory.


I have also tested letro vs aromastin and adex. The only difference ive found is that letro's e2 suppression is liner with once daily doses. Adex would require 2x per day and aromastin 3-4 x per day.

Most people think its stronger. It only gets ride of the spikes the others leave.

Letro can cross into other tissue and fight estrogen. This can be good or bad. Its great for going in to fat tissue. But it can cross into the brain as well and cause libido problems.
 
Aside from lowering E2 don't all AI's lower IGF levels? I read this recently and don't know if it's true. If it is I'm gonna keep the test low enough in all my cycles to where I don't need an AI-which would be like 210mg of Prop which brings my E2 into the 40's and then add some Masteron or Proviron to free up some Test and make that low dose more effective and add a hefty dose of a nice clean anabolic like Primo or DHB or NPP to make up the difference.
 
Adex has been shown to reduce igf. But I think you are thinking serms. Like nolva. Estrogen is needed for the liver to make igf. Since serms are not real estrogen they can't do the job.
 
What always threw me off about your tests Kaladryn is the presence of tren hex in your cycle. How do those effect your e2 values and what are your true e2 values if the test is flawed cause of the tren. Are you actually somewhere near 20-40 e2 and your hex is inflating the values 75+ for example?

Good point pieguy. I hadn't realized that was a component of his cycles. How much Hex was being run?


As a side note, Id like to know all about your experiences and opinions with tren hex Kaladryn.

Yes the tren could have been elevating the values, however doses of tren were consistant on each test, 350mg. This is the most I have run, and just for contest prep purposes. I notice huge strength increases (almost too much strength, injury inducing), metabolism increase, and absolutely zero side effects other than prolactin related gyno that was controlled with .5mg prami (no gyno if prami is run preventively).

I don't mess with tren anymore, but if I did, I'd probably just use acetate. There is no difference between esters except halflife. This may be obvious, but the myths persist...
 
Isn't the point of a cruise to give your body a break? I read most of Dr. G's posts, I disagree with almost everything he says.

Kaladryn, you disagree with almost "everything" Dr.G says? That's pretty harsh considering his credentials, but to each his own...

I've been cruising on test e/tren e at 100/100mg week for the last 4 weeks and have had nothing but good results. Sides are near non-existent and I've actually gained mass. I've also increased my intensity in the gym, so that obviously may be part of it, but needless to say I've been very happy with this protocol.

Much respect,
 

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