Dr. Scally is a qualified endocrinologist (yes I know he was struck off in Texas) with thousands of patients being treated in his very long career. He's seen the BW of certain drugs, protocols and dosages. Yet, you think you know better. Pretty amazing to be honest but anyway.
Why do you think the metabolites of Tamoxifen and Clomid are both at work 6 weeks AFTER cessation? Even if they are, its just a theory of yours they're active enough to impact the negative feedback mechanism.
Tapering SERMs or using an AI towards the end of the PCT protocol will mitigate the negative effects estrogen is going to cause later on.
What data do you have the states that SERMs cause shutdown after usage when treating hypogonadism?
I'm not questing his doctoral degree, being a doctor doesn't excuse oneself from needing real research to prove things. When a team of doctors gets together, and they study one thing, and do tests on it, and come to a conclusion, and then other doctors look at what they did and review it, THAT knows better than some individual doctor who can't control his data properly.
I've posted studies on the clomid metabolites years ago, I've also done clomid and seen the bloodwork, it's only clomid, it has one metabolite that has an 8 day halflife or something like that. Almost everyone who uses clomid gets bloodwork done a month after stopping and they have very high test levels, try two months after...
I totally agree with using an AI during the end of a cycle to help with estrogen dominance, that IS 'pct' to me, but it's the "restarting" I don't believe has anything to it.
SERMS block negative E2 feedback at the hyperthamous and stimulate GNRH>LH>T production, this raises E2 through aromatization, but the higher E2 doesn't shut down GNRH production because it's being blocked by the SERM. As soon as you stop the SERM, and even if you taper, you are going to have a state of higher E2 that shuts off GNRH and LH/T, at least to some extent. AIs does the same thing in a different way, they stimulate test by lowering E2, as soon as you remove the AI, E2 climbs, GNRH/LH/T all go down. Of course, eventually, low T will lead to low E2 which will stimulate GNRH again, assuming one doesn't have some kind of "hyperactive aromatase" state, which has been proposed by an endo but is theoretical.