Lactulose is a synthetic sugar used to treat constipation. It is broken down in the colon into products that pull water out from the body and into the colon. This water softens stools.
Lactulose is also used to reduce the amount of ammonia in the blood of patients with liver disease.
https://www.google.com/search?q=lac...=chrome..69i57j69i59&sourceid=chrome&ie=UTF-8
Not saying you have liver disease but you need to have a workup including imaging to cover all of your bases.
Read this and check out the link...
"Primary causes of hyperammonemia include congenital enzymopathies in the urea cycle, such as deficiencies of ornithine transcarbamoylase and argininosuccinate lyase. These disorders can lead to varying degrees of hyperammonemia depending on the enzyme affected and on whether the genetic deficiency is heterozygous or homozygous. Secondary hyperammonemia occurs commonly in the presence of hepatic disorders leading to portosystemic encephalopathy, but can occur in the absence of hepatic dysfunction in disorders like Reye's syndrome, ureterosigmoidostomy, and infection in a neurogenic bladder. Drug toxicity as a result of disruption of mitochondrial pathways by drugs like cyanide, carbamazepine, valproic acid, iron, and cytotoxics can also cause secondary hyperammonemia, and this is thought to be the main mechanism by which nonhepatic or noncirrhotic hyperammonemia can occur in people exposed to these drugs (2, 4–9)."
"Noncirrhotic hyperammonemia as a cause of altered mental status remains a diagnosis of exclusion. In our patient, the elevated blood ammonia level and the rapid resolution of symptoms coupled with the reduction of his blood ammonia level led us to suspect that hyperammonemia may have accounted for his presentation. His clinical presentation and subsequent clinical course were similar to those of patients described in prior case reports of noncirrhotic/nonhepatic hyperammonemia (2–8)."
"The diagnosis of hyperammonemia may be challenging and requires a high index of suspicion. However, it should be considered when a clear cause of altered mental status is not obvious after basic investigations. As an initial step, it is advisable to exclude hypo- or hyperglycemia, azotemia, liver failure, electrolyte imbalance, sepsis, structural and vascular pathologies like stroke, and central nervous system involvement by cancers. Thereafter, the evaluation can be expanded to consider less common etiologies."
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4569228/