I'm wondering if anyone here has information about T3 supplementation? I do not want to suppress my t3 production, just supplement it while taking peptides. I've read 12.5mcg's/day is a supplement dose, but with no evidence or proof behind it.
In that case dont even use the T3 at all. Use supplements like Fenugreek, Guggulsterones, Kelp etc.
dont be one of those guys thats a slave to t3 as an excuse to eat like a moron. small doses of it (12.5-50mcg) will improve protein synthesis. i use it @ 50mcg 1 month on/1 off without any ramping and see no problems with feeling run down. my bloodwork does show higher than normal t3 levels tho. but thats a no brainer
why is this even necessary? i use gh with no supplementation at all to t3 to t4................is this bad?
well, i wont touch slin, would adding t3 be worth it ya think on 4ius 5/2?
According to what?small doses of it (12.5-50mcg) will improve protein synthesis.
For starters, how about the fact that T3 is notoriously catabolic? This is well established at higher doses. As far as I know, it hasn't been examined at lower doses, so you're rolling the dice.if u have t3 tabs or can get em, why not?
if u have t3 tabs or can get em, why not? more protein synthesis is never a bad thing. however one draw back i do find is that when i do cycle my t3 its hard to monitor my calorie intake. of course u sweat like u never have before and for that month on i have to eat like Man vs. Food to continue going up on the scale. offseasons great for that tho! not saying whlie ur on t3 its smart to put the buffet outa business tho. i do every once in a while tho. if u do, take a shot of igf-1 prior
its not bad, t3 at low doses just adds to the benefits. greater protein synthesis. its just the basics to administering gh for growth. gh+slin+t3 = size
im interseted in why you would do this before a buffet type meal? not being a smartass just genuinely interested if its beneficial
According to what? **broken link removed** sure doesn't support that. The researchers found that IGF-1 was more potent than insulin at stimulating proadipocyte proliferation and differentiation (creating new fat cells). It also found that IGF-1 was as potent as insulin at stimulating glucose uptake into adipocytes (fat cells) and as potent as insulin at inhibiting lipolysis (preventing fat release).igf is a major nutrient shuttling agent without the fat storing of insulin.
According to what? **broken link removed** sure doesn't support that. The researchers found that IGF-1 was more potent than insulin at stimulating proadipocyte proliferation and differentiation (creating new fat cells). It also found that IGF-1 was as potent as insulin at stimulating glucose uptake into adipocytes (fat cells) and as potent as insulin at inhibiting lipolysis (preventing fat release).
To quote the paper:
- "IGF-I was equipotent with insulin...in promoting glucose uptake in mature adipocytes"
- "Insulin (20 nM) inhibited the isoproterenol-stimulated lipolysis to control values (Fig. 5A). IGF-I, also at 20 nM, was equipotent with insulin in inhibiting the increased lipolysis"
- "This study demonstrates that in 3T3-L1 cells in vitro, IGF-I, rather than insulin, is more likely to be the main regulator of preadipocyte proliferation."
- "IGF-I is markedly more potent than insulin in stimulating the clonal expansion of 3T3-L1 preadipocytes prior to differentiation into mature adipocytes."
- "In mature adipocytes, we found that IGF-I is equipotent with insulin in promoting glucose uptake"
- "Importantly, we have also shown that IGF-I is equipotent with insulin at inhibiting isoproterenol-stimulated lipolysis. Both insulin and IGF-I inhibited lipolysis with an EC50 of 15 nM. IGFs may therefore play a role in the regulation of lipolysis and free fatty acid secretion in adipocytes."