PHIL HERNON
Banned
- Joined
- Jun 6, 2002
- Messages
- 14,932
I recieved this letter from my past training partner and great friend, David Villegas........I cherish all info from him and learned a TON from him when I was preparing for the USA in 1995
Phil,
Here is food for thought.
One of the mainstays of the training by genotype was the volume of intensity and rest. If you rememer the type A training was fairly low compared to other blood types. Up to that point the only reesarch available was from a small laboratory in england and the research we did at UNM.
As you know there has been a controversy as to how muscle growth really works. Words like Hyperplasia, hypertrophy and a combination of the two caused debates and major dessention within the sports community. Well there has been a light at the end of the tunnel. The only way to help piece the puzzle is to perform strict muscle biopsies with radio labels or chemicals in a working muscle to find out how it really functions.
A research facility in Iceland working specifically in genentics and human performance did such an experiement about 2 years ago on healthy muscular men who had significant weight lifting experience. Something you rarely find in todays exercise physiology experiments.
Muscle biopsies were done on all men and the ratio of type 1 and type 2 muscle fibers were measured. After the experiment was completed more biopsies were done in order to measure how exactly growth was taken place. Obviously I am condensing the results to prove a single point. What they found out was that hyperplasia was not a factor in growth and hypertrophy played only a minute role. What astounded them was that the body "recruited" type 2 muscle fibers and chemically, physiology converted them to type 1 muscle fibers with a small increase in the myosin fiber. Apparently the body has the ability to change itself structurally to adapt to its environment. Here is the interesting conclusion. Those with type A or O blood have a propensity to have a larger amount of type 2 muscle fiber which provides the body with a "sink" of available muscle tissue for conversion. The difference of muscle fibers (type2) ranges between a 7-13% larger portion between A,O to B,AB. The training protocol used was 3 sets to failure with a 2 minute rest utilizing 85% of 1RM.
This company in Iceland (Pharmacogenetics) is doing some amazing research. They are working on gene doping with athletes and I am suppsed to do a site rotation in June of this year.
Phil,
Here is food for thought.
One of the mainstays of the training by genotype was the volume of intensity and rest. If you rememer the type A training was fairly low compared to other blood types. Up to that point the only reesarch available was from a small laboratory in england and the research we did at UNM.
As you know there has been a controversy as to how muscle growth really works. Words like Hyperplasia, hypertrophy and a combination of the two caused debates and major dessention within the sports community. Well there has been a light at the end of the tunnel. The only way to help piece the puzzle is to perform strict muscle biopsies with radio labels or chemicals in a working muscle to find out how it really functions.
A research facility in Iceland working specifically in genentics and human performance did such an experiement about 2 years ago on healthy muscular men who had significant weight lifting experience. Something you rarely find in todays exercise physiology experiments.
Muscle biopsies were done on all men and the ratio of type 1 and type 2 muscle fibers were measured. After the experiment was completed more biopsies were done in order to measure how exactly growth was taken place. Obviously I am condensing the results to prove a single point. What they found out was that hyperplasia was not a factor in growth and hypertrophy played only a minute role. What astounded them was that the body "recruited" type 2 muscle fibers and chemically, physiology converted them to type 1 muscle fibers with a small increase in the myosin fiber. Apparently the body has the ability to change itself structurally to adapt to its environment. Here is the interesting conclusion. Those with type A or O blood have a propensity to have a larger amount of type 2 muscle fiber which provides the body with a "sink" of available muscle tissue for conversion. The difference of muscle fibers (type2) ranges between a 7-13% larger portion between A,O to B,AB. The training protocol used was 3 sets to failure with a 2 minute rest utilizing 85% of 1RM.
This company in Iceland (Pharmacogenetics) is doing some amazing research. They are working on gene doping with athletes and I am suppsed to do a site rotation in June of this year.