I ran quite a bit of test and tren with trest and noticed my results were better than the guys who ran strictly trest only (there were like 20 trest logs lol).
Do you buy into the theory of shoulders and traps having the most androgen receptors in our bodies Mike? Because I know when I ran trest my traps exploded with growth and thats without doing anything extrta for them....im no expert but I assumed the extremely high androgen profile of the trest was what caused it? Unfortunately when I ran out trest they went back to normal lol
As far as I am aware the traps do have a higher AR density than other muscles, but I didn't read the studies myself so I suppose I could be wrong. However, considering the number and type of sources I've heard this from, I've always just assumed it was a fact.
But...I think you may be drawing a non-existent link between AR density and a steroid's androgenic rating. Just because a steroid has a higher androgenic rating, it does not necessarily mean it will cause more growth in AR dense muscles compared to other steroids.
You see, the term "androgenic rating" refers ONLY to a steroid's androgenic potency in prostate tissue. Many steroid users are under the false impression that a steroid's AR rating is a reference to its general androgenic character throughout the body, but this is not the case. A steroid's androgenic potency can vary tremendously from tissue to tissue, which is why a steroid can cause strong androgenic side effects in one area, but not in another. A good example would be Mast. Mast is fairly easy on the prostate compared to many other steroids, but is known to be rather harsh on the hairline. If a steroid's androgenic rating represented its overall androgenic character, Mast would be just as easy on the hairline as it is on the prostate, yet it's not. The same can be said of Winstrol and many other steroids.
The point here is that androgenic ratings are absolutely worthless for determining how a steroid might act in non-prostate tissues. As a result, they also have no value when it comes to ascertaining a steroid's muscle building potency.
Moving on. The term "androgen receptor" refers to what we might call a "docking port" for AAS. Androgen receptors are found in many different tissues all throughout the body and are the main pathway through which steroid's exert almost all of their effects--both anabolic and androgenic. If a steroid doesn't attach to an androgen receptor, it will pretty much just float around doing nothing. The bottom line is that AAS need androgen receptors in order exert their effects in bodily tissues, regardless of whether we are talking about muscles, vocal chords, hair follicles, skin, prostate, etc.
So, just because trestolone has a high androgenic rating, it does not mean it is going to have a greater effect in the traps than other muscles. It simply means that the traps have more available "docking ports" for AAS in general...regardless of the type. To put it another way, AR dense muscles do not respond preferentially to steroid's with high androgenic ratings. There is simply no connection between a steroid's androgenic rating and its ability to stimulate muscle growth in AR dense tissues. AR dense muscles respond better to "ALL" steroids.
Now, if your traps grew more than your other muscles during your trestolone cycle, it was only because that particular steroid worked well for you, not because the traps responded selectively to trestolone. Sometimes one bodypart just grows faster than others, for various reasons.