- Joined
- Sep 29, 2008
- Messages
- 714
In regards to stomach enlargement. A lot of that is actually visceral fat around the organs. The abs can be seen of course because subq fat is low so don't be fooled.
People think the intestinal lining is growing half a foot in thickness? Organs aren't growing that large.
High dosages of testosterone and longer duration can influence visceral fat growth.
Although some studies show testosterone reduces visceral fat you have to take into consideration the dosage and duration. An opposite effect can occur given the above circumstances.
Keep in mind that GH releases fatty acids into the bloodstream. Insulin can deposit fatty acids back into adipose tissue. If both are active at the same time (remember those protocols?) I can see how fat can be redistributed in the body rather than being expended as energy.
Also consider what can cause cortisol increases long term and short term.
Here is a quote on cortisol:
"Cortisol directly effects fat storage and weight gain in stressed individuals. Tissue cortisol concentrations are controlled by a specific enzyme that converts inactive cortisone to active cortisol (9-11). This particular enzyme is located in adipose (fat) tissues. Studies with human visceral (fat surrounding the stomach and intestines) and subcutaneous fat tissue have demonstrated that the gene for this enzyme is expressed more by obese conditions (11). It has also been demonstrated in research that human visceral fat cells have more of these enzymes compared to subcutaneous fat cells. Thus, higher levels of these enzymes in these deep fat cells surrounding the abdomen may lead to obesity due to greater amounts of cortisol being produced at the tissue level. As well, deep abdominal fat has greater blood flow and four times more cortisol receptors compared to subcutaneous fat (8). This may also increase cortisol’s fat accumulating and fat cell size enlarging effect."
Just some thoughts.
People think the intestinal lining is growing half a foot in thickness? Organs aren't growing that large.
High dosages of testosterone and longer duration can influence visceral fat growth.
Although some studies show testosterone reduces visceral fat you have to take into consideration the dosage and duration. An opposite effect can occur given the above circumstances.
Keep in mind that GH releases fatty acids into the bloodstream. Insulin can deposit fatty acids back into adipose tissue. If both are active at the same time (remember those protocols?) I can see how fat can be redistributed in the body rather than being expended as energy.
Also consider what can cause cortisol increases long term and short term.
Here is a quote on cortisol:
"Cortisol directly effects fat storage and weight gain in stressed individuals. Tissue cortisol concentrations are controlled by a specific enzyme that converts inactive cortisone to active cortisol (9-11). This particular enzyme is located in adipose (fat) tissues. Studies with human visceral (fat surrounding the stomach and intestines) and subcutaneous fat tissue have demonstrated that the gene for this enzyme is expressed more by obese conditions (11). It has also been demonstrated in research that human visceral fat cells have more of these enzymes compared to subcutaneous fat cells. Thus, higher levels of these enzymes in these deep fat cells surrounding the abdomen may lead to obesity due to greater amounts of cortisol being produced at the tissue level. As well, deep abdominal fat has greater blood flow and four times more cortisol receptors compared to subcutaneous fat (8). This may also increase cortisol’s fat accumulating and fat cell size enlarging effect."
Just some thoughts.