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HCG for Restoring Test Level

BigMatt

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What would Happen If A Natural has Killed his Test Level With Doing INTENSE CARDIO on a Caloric Restrited Diet.

Then he Supplement With HCG.

Would that help?

What would a Doctor prescibe to a Male for Restoring his T Levels Up in Optimal Range?
 
From what I've read hcg suppresses endogenous testosterone production (taken from swale's PCT protocol). I don't know everything about thos topic, but I'm pretty sure that decreased t levels from suppression by aas are caused by different things than intense cardio, and therefore have different treatments. I recently spoke with my HRT about substituting HCG for test, and they said this was fine due to the nature of my deficiency. I personally dealt with low t levels for years because I was an endurance cyclist (>200 miles per week), and I tried all sorts of things to boost my natural test levels. What I didn't want to do, though, was suppress my natural ability to produce test. Good luck
 
From what I've read hcg suppresses endogenous testosterone production (taken from swale's PCT protocol). I don't know everything about thos topic, but I'm pretty sure that decreased t levels from suppression by aas are caused by different things than intense cardio, and therefore have different treatments. I recently spoke with my HRT about substituting HCG for test, and they said this was fine due to the nature of my deficiency. I personally dealt with low t levels for years because I was an endurance cyclist (>200 miles per week), and I tried all sorts of things to boost my natural test levels. What I didn't want to do, though, was suppress my natural ability to produce test. Good luck

I thought it mimics LH which in turn signals the testes to produce test. I would assume what get suppressed is the natural production of LH. What I don't know is how soon that comes back and in what capacity after ceasing use of HCG.
 
HCG is a LH mimetic

Check free thyroid levels, I think this has something to do with this, as may prolactin.

It will increase testosterone, but in higher doses be careful of estradiol.

It causes suppression not in the testis but at the pituitary/hypothalmus level. Low doses with a serm or anti-e would produce a noticible difference in a lot of men.

Test enanthate or cypionate are two common approaches as well as ansrogel. (Proviron as well in Europe.)
 
From what I've read hcg suppresses endogenous testosterone production (taken from swale's PCT protocol).
no it causes production of endogenous testosterone but will suppress LH/FSH production
 
no it causes production of endogenous testosterone but will suppress LH/FSH production

From swale's protocol:

"I want my patients to stop taking HCG within a week after the end of the cycle. The testosterone production it induces will further inhibit recovery, as will using Androgel, or any other testosterone preparation, while in recovery. There is no escaping this, as there is no such thing as a "bridge". Just because you are not inhibiting the HPTA for the entire 24 hours does not mean you are not suppressing it at all. IOW, you can't "fool" the body? it is smarter than you are."

So swale is saying that HCG induces testosterone production, but inhibits recovery. I thought that "inhibiting recovery" would mean that hcg is suppressing natural testosterone production. Isn't that what recovery is supposed to be about? But let me digress, LH/FSH production causes production of testosterone, yes? Then if LH/FSH is suppressed, then it also suppresses natural testosterone production? The leydig cells are directly stimilated by HCG to produce testosterone, right? I'm a little lost. I would dig out my anatomy and physiology book, but loaned it to a friend taking the class. I'm trying to learn about this, maybe you'd care to share more? Maybe hmg (or whatever the new expensive drug's name is) is the way to go.
 
no it causes production of endogenous testosterone but will suppress LH/FSH production


I had thought that the advantage of taking HCG was that the body restores LH/FSH faster than the testes respond to endogenous gonadotropins & that giving the Leydig cells a head start on endogenous LH/FSH was more urgent than waiting for LH/FSH to return to normal.

I think that's right anyway. Anyone remember any study evidence to support or dismiss this?

I'd hate to think that we're all poking female fertility drugs because some pharmaceutical salesman with a connection in the BBing community decided that having big fluffy nuts after a cycle was more important than stimulating the return of endogenous LH & FSH!!!

(Wouldn't we all be suckers then!)
 
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