Good look here, very balanced view of the subject. Talks about how there is limited scientific evidence linking AAS directly to heart problems. It is the nature of the issue that makes it hard to have a true scientific study. I wonder if anyone has run a good study on rats. Id like to see that.
the study:
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3827559/
This is like what happend to me. From this study:
"The risk of sudden death from cardiovascular complications in the athlete consuming anabolic steroids can occur in the absence of atherosclerosis. Thrombus formation has been reported in several case studies of bodybuilders self-administering anabolic steroids (Ferenchick, 1991; Fineschi et al., 2001; McCarthy et al., 2000; Sahraian et al., 2004). Melchert and Welder, 1995 have suggested that the use of 17α-alkylated steroids (primarily from oral ingestion) likely present the highest risk for thrombus formation. They hypothesized that anabolic steroid consumption can elevate platelet aggregation, possibly through an increase in platelet production of thromboxane A 2 and/or decreasing platelet production of prostaglandin PgI 2, resulting in a hypercoagulable state."
I had no atherosclerosis.
Just added this, looks like rat studies have taken place and show LV hypertrophy and heart problems:
"Left ventricular function and anabolic steroid use/abuse has been examined. Climstein and colleagues (2003) demonstrated that highly strength-trained athletes, with no history of anabolic steroid use exhibited a higher incidence of wave form abnormalities relative to recreationally-trained or sedentary individuals. However, when these athletes self-administered anabolic steroids, a higher percentage of wave form abnormalities were exhibited. Further evidence suggestive of left ventricular dysfunction has been reported in rodent models. A study on rats has shown that 8 weeks of testosterone administration increased left ventricle stiffness and caused a reduction in stroke volume and cardiac performance" It goes on to explain some problems with this study however.
You had no atherosclerosis but you did have some plaque build up, no?
And I remember you said your Hematocrit was around 62 in the months before the attack but got it down to a better range by donating although there’s been things posted showing evidence that a hematocrit rebound can occur when donating (someone correct me if I’m wrong on that) and do you remember what your platelet count was? Since the first paragraph talks about platelets increasing and causing a “hypercoagulable state.”