You can maintain calories by eating caloric dense foods like peanut butter. But eating too much your kinda defeating the purpose due to the higher fat content. Eat a wholesome diet high in protein: Keep fueling your body with high quality food. It will help your body function properly and maintain a healthy status. Protein is your best friend when maintaining muscle mass. Eating 1.5-2.0 grams of protein per kg of body weight is a great general guideline. Here are some high protein foods you can eat:. So you want to maintain a high caloric intake with quality protein and good carbs.Can be either of the two or both at the same time.
All but if your androgens are right and you’re eating enough food and lots of protein T3 can actually speed up anabolism.Too much T3? Too little calories/protein while using T3? Or a combination of both?
I have read this as well.All but if your androgens are right and you’re eating enough food and lots of protein T3 can actually speed up anabolism.
But yet damn near every competitive bodybuilder uses t-3 in their prep. I guess that goes to show the true power of anabolic steroids.The notion that T3 benefits protein turnover comes from L. Rea's Building the Perfect Beast, a work of science fiction. While there is a slight increase in protein synthesis (9% at 100mcg T3 daily x 2 weeks) the increase in protein catabolism/breakdown is greater (-12% & 24% Leucine oxidation) meaning net protein catabolism/breakdown.
Most alarmingly, for the minor benefit on energy expenditure/RMR of 10-15%, T3 preferentially catabolizes muscle tissue. Specifically, Type IIA fibers (those that reflect trained/hypertrophied muscle) shrunk in cross-sectional area by 15% in 6 weeks of T4+T3 use.
The notion that T3 benefits protein turnover comes from L. Rea's Building the Perfect Beast, a work of science fiction. While there is a slight increase in protein synthesis (9% at 100mcg T3 daily x 2 weeks) the increase in protein catabolism/breakdown is greater (-12% & 24% Leucine oxidation) meaning net protein catabolism/breakdown.
Most alarmingly, for the minor benefit on energy expenditure/RMR of 10-15%, T3 preferentially catabolizes muscle tissue. Specifically, Type IIA fibers (those that reflect trained/hypertrophied muscle) shrunk in cross-sectional area by 15% in 6 weeks of T4+T3 use.
But yet damn near every competitive bodybuilder uses t-3 in their prep. I guess that goes to show the true power of anabolic steroids.
True that androgens seem to spare some skeletal muscle (not surprising as they are anticatabolic in muscle). Unfortunately there's no good data on to what extent. I do hope you recognize the weakness of the argument that "damn near every competitive bodybuilder" doing something does not actually make it the best choice. What if doing something differently actually leads to better results?But yet damn near every competitive bodybuilder uses t-3 in their prep. I guess that goes to show the true power of anabolic steroids.
I was just giving a point in that if the muscle loss was to any significant extent bodybuilders at the highest level would not use it, yet 90%+ use it as a tool to get shredded. Maybe the argument is in the dosage thus keeping dosage below certain levels. Although a shit load of high level female bodybuilders seem to use pretty high amounts of it.True that androgens seem to spare some skeletal muscle (not surprising as they are anticatabolic in muscle). Unfortunately there's no good data on to what extent. I do hope you recognize the weakness of the argument that "damn near every competitive bodybuilder" doing something does not actually make it the best choice. What if doing something differently actually leads to better results?
Also, a correction: the -15% change in Type IIA fiber cross-sectional area was for 100mcg daily T3 for 2 weeks. Interestingly, from all the evidence, between 2-3 weeks there is an inflection point in the catabolism of muscle and some mechanism seems to spare FURTHER muscle loss while fat loss continues.
Gotcha. I just note that some people succeed in spite of what they do, not because of it, and by definition most competitive bodybuilders do not actually win/place/show. I personally think T3 has an unfavorable risk-reward profile. That's a good point about dosage, from what I've seen if I absolutely had to use T3 (IDK, say availability or something), I wouldn't go over 75 mcg daily and I'd make sure to use it with the right androgens. I also wonder whether some attributes of the different female categories might be OK with some muscle loss (doesn't seem the most rational thing to me to ever catabolize muscle)... but perhaps a wellness or bikini competitor might be able to justify some muscle loss at the last minute for some necessary fat loss.I was just giving a point in that if the muscle loss was to any significant extent bodybuilders at the highest level would not use it, yet 90%+ use it as a tool to get shredded. Maybe the argument is in the dosage thus keeping dosage below certain levels. Although a shit load of high level female bodybuilders seem to use pretty high amounts of it.
My post was not saying the science is wrong, im just saying real world applications in bodybuilders using high levels of PED's seem to tell a different story.
If your question is whether there is any study in humans on clen looking at the prevention of muscle atrophy in man with caloric restriction, the answer is no (unless there's data I haven't seen; but doubtful). But we do know it mitigates protein degradation mechanistically. It is demonstrably skeletal muscle anabolic in man, increases sprint performance, strength, is lipolytic, and increases skeletal muscle mass while decreasing fat mass. It's a potent recomp agent. I've written about it somewhat extensively here: Clenbuterol focus: Research on Clen & Beta2- Adrenergic Compounds, Protocols@Type-IIx , is there any research supporting clen being anti catabolic in humans? I know animal models have shown anti catabolic activity. I think it might of been horses??
If your question is whether there is any study in humans on clen looking at the prevention of muscle atrophy in man with caloric restriction, the answer is no (unless there's data I haven't seen; but doubtful). But we do know it mitigates protein degradation mechanistically. It is demonstrably skeletal muscle anabolic in man, increases sprint performance, strength, is lipolytic, and increases skeletal muscle mass while decreasing fat mass. It's a potent recomp agent. I've written about it somewhat extensively here: Clenbuterol focus: Research on Clen & Beta2- Adrenergic Compounds, Protocols
If your question is whether there is any study in humans on clen looking at the prevention of muscle atrophy in man with caloric restriction, the answer is no (unless there's data I haven't seen; but doubtful). But we do know it mitigates protein degradation mechanistically. It is demonstrably skeletal muscle anabolic in man, increases sprint performance, strength, is lipolytic, and increases skeletal muscle mass while decreasing fat mass. It's a potent recomp agent. I've written about it somewhat extensively here: Clenbuterol focus: Research on Clen & Beta2- Adrenergic Compounds, Protocols