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Ester Q????

Showy0023

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Is aborption time always dependent on ester type? For example would Tren E and Test E become active in the bloodstream in the same amount of time? Or does compound type play a role?

Thanks,

Showy
 
I would assume the compound plays a bit of a role too...

Just look at the half lives of dbol and winny.

Those are oral though...
 
Is aborption time always dependent on ester type? For example would Tren E and Test E become active in the bloodstream in the same amount of time? Or does compound type play a role?

Thanks,

Showy

the ester is what the body must 'break off' the parent molecule to use it. since the ester is the same, the rates of enzymatic breakdown are the same.

now after the parent molecule is detached, the rate soley depends on the properties of the substance you are using

overall the ester is what determines time, go by that
 
the ester is what the body must 'break off' the parent molecule to use it. since the ester is the same, the rates of enzymatic breakdown are the same.
I'd just point out that longer esters are not enzymatically harder to "break off". That's not what causes the longer half-life. Rather, longer esters are more lipophillic, so they remain in the injection depot longer. They're less likely to partition into the aqueous interstitual fluid around the injection site. Once they leave the depot though, the ester is quickly removed by esterases. The rate of that enzymatic breakdown is rapid regardless of the ester length.
 
^ interesting! man I learn something cool from almost every one of your posts.
 
Thanks guys.
 
Oil will slow the absorption, anything in water will enter the bloodstream quicker. Usually hurts more too, as the water is the first to leave and sometimes the hormone stays behind in the muscle causing discomfort
 

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