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- Apr 9, 2021
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Do you mean, what are doses of rhGH that induce hyperglycemia & of Tren that pose particular cardiac harms ?What dose are you thinking here?
Tren poses these particular (meaning > the average AAS) harms (MR antagonism, corticosteroid derangement) at all doses, almost certainly dose-dependently. So as doses ramp up, so too does risk of a fatal or nonfatal acute coronary event. This risk is multifactorial, and depends on factors like past AAS use history, genetic proclivity (heritable factors), diet (e.g., saturated fat intakes), activity, etc.
RhGH (a term to distinguish it from endogenous GH; but now, also to irritate BiggerP) also directly stimulates insulin resistance within 1 - 2 h, such that doses > replacement (rhGH suppresses GH secretion for ~ 1 day, +/-, with some escape from suppression that differs between individuals) dose-dependently contribute to the risk of hyperglycemia (as doses ramp up, so too does the risk of blood glucose > 130 mg/dL or 7.2 mmol/L after fasting for at least 8 h, defined as fasting hyperglycemia). The consequences of this depends on multiple factors like body fat mass, diet, etc.