I
believe this but cannot provide evidence for it. While it conforms with my experience, there's just been no data on Tren in this regard (wrt hemodynamic changes).
One confounding factor to this, however, is an important one: we're virtually all using mega-doses of Trenbolone Acetate. I've posted in reference to this before - see:
https://www.professionalmuscle.com/forums/index.php?threads/just-a-bit-of-tren.170908/post-3077660 for a discussion of what "low dose Tren" might actually look like.
Suffice it to say, the 350 mg tren ace first use principle is EXTREMELY high dose. It is quite possible that if we saw a shift to saner doses of Tren in line with its relative potency, that it may not be a markedly potent hematinic agent.
* i.e., trenbolone is a ~3X more potent AR agonist than testosterone; so 350 mg tren ace is, at least, equivalent to ~1,050 mg of testosterone enanthate just in terms of genomic action via the AR (it's actually a lot more potent than this in reality, because it serves to modulate glucocorticoids and works via other pathways than classical genomic action).